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Department of Genetics
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Drosophila synaptotagmin I null mutants show severe alterations in vesicle populations but calcium-binding motif mutants do not.
The Journal of comparative neurology May, 2006 | Pubmed ID: 16528727
C2B polylysine motif of synaptotagmin facilitates a Ca2+-independent stage of synaptic vesicle priming in vivo.
Molecular biology of the cell Dec, 2006 | Pubmed ID: 16987956
Lysosomal dysfunction promotes cleavage and neurotoxicity of tau in vivo.
PLoS genetics Jul, 2010 | Pubmed ID: 20664788
The unfolded protein response protects from tau neurotoxicity in vivo.
PloS one , 2010 | Pubmed ID: 20927324
A Drosophila model of closed head traumatic brain injury.
Proceedings of the National Academy of Sciences of the United States of America Oct, 2013 | Pubmed ID: 24127584
University of Wisconsin-Madison
Rebeccah J. Katzenberger1,
Carin A. Loewen2,
R. Tayler Bockstruck1,
Mikal A. Woods3,
Barry Ganetzky2,
David A. Wassarman1
1Department of Cell and Regenerative Biology, University of Wisconsin-Madison,
2Department of Genetics, University of Wisconsin-Madison,
3Department of Natural Sciences, University of Puerto Rico-Aguadilla
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