Satoru Yamagishi

Brain-derived neurotrophic factor-induced potentiation of Ca(2+) oscillations in developing cortical neurons.

Apoptosis-signal regulating kinase-1 is involved in the low potassium-induced activation of p38 mitogen-activated protein kinase and c-Jun in cultured cerebellar granule neurons.

Comparison of inhibitory effects of brain-derived neurotrophic factor and insulin-like growth factor on low potassium-induced apoptosis and activation of p38 MAPK and c-Jun in cultured cerebellar granule neurons.

Involvement of caspase-4 in endoplasmic reticulum stress-induced apoptosis and Abeta-induced cell death.

JAB1 participates in unfolded protein responses by association and dissociation with IRE1.

Promotion of axon regeneration by myelin-associated glycoprotein and Nogo through divergent signals downstream of Gi/G.

P311 accelerates nerve regeneration of the axotomized facial nerve.

p75 neurotrophin receptor signaling in the nervous system.

Biological activity of neurotrophins is dependent on recruitment of Rac1 to lipid rafts.

ERK1/2 are involved in low potassium-induced apoptotic signaling downstream of ASK1-p38 MAPK pathway in cultured cerebellar granule neurons.

Diverse functions of the p75 neurotrophin receptor.

Wallerian degeneration involves Rho/Rho-kinase signaling.

[Inhibition of axon regeneration by myelin inhibitors].

Brain-derived neurotrophic factor-induced potentiation of glutamate and GABA release: different dependency on signaling pathways and neuronal activity.

Multiple signals regulate axon regeneration through the Nogo receptor complex.

RGMa inhibition promotes axonal growth and recovery after spinal cord injury.

Myelin-associated glycoprotein inhibits microtubule assembly by a Rho-kinase-dependent mechanism.

An in vitro model for Lewy body-like hyaline inclusion/astrocytic hyaline inclusion: induction by ER stress with an ALS-linked SOD1 mutation.

Genetic ablation of FLRT3 reveals a novel morphogenetic function for the anterior visceral endoderm in suppressing mesoderm differentiation.

Familial amyotrophic lateral sclerosis (FALS)-linked SOD1 mutation accelerates neuronal cell death by activating cleavage of caspase-4 under ER stress in an in vitro model of FALS.

FLRT2 and FLRT3 act as repulsive guidance cues for Unc5-positive neurons.

[FLRT family proteins act as repulsive guidance cues for Unc5-positive neurons].

Netrin-5 is highly expressed in neurogenic regions of the adult brain.

Ion channels, guidance molecules, intracellular signaling and transcription factors regulating nervous and vascular system development.