Maciej Markiewski

Increased C5a receptor expression in sepsis.

The proinflammatory mediators C3a and C5a are essential for liver regeneration.

Novel monoclonal antibodies against mouse C3 interfering with complement activation: description of fine specificity and applications to various immunoassays.

C3a and C3b activation products of the third component of complement (C3) are critical for normal liver recovery after toxic injury.

Partial hepatectomy induced liver proteome changes in mice.

Liver inflammation and regeneration: two distinct biological phenomena or parallel pathophysiologic processes?

A high-fat diet impairs liver regeneration in C57BL/6 mice through overexpression of the NF-kappaB inhibitor, IkappaBalpha.

Liver regeneration: a link to inflammation through complement.

Complement and coagulation: strangers or partners in crime?

The role of complement in inflammatory diseases from behind the scenes into the spotlight.

C5a and TNF-alpha up-regulate the expression of tissue factor in intra-alveolar neutrophils of patients with the acute respiratory distress syndrome.

Complexity of complement activation in sepsis.

Modulation of the antitumor immune response by complement.

Cutting edge: members of the Staphylococcus aureus extracellular fibrinogen-binding protein family inhibit the interaction of C3d with complement receptor 2.

The regulation of liver cell survival by complement.

Is complement good or bad for cancer patients? A new perspective on an old dilemma.

Diet-induced hepatocellular carcinoma in genetically predisposed mice.

Unwelcome complement.

Complement anaphylatoxin C5a contributes to hemodialysis-associated thrombosis.

C5a receptor enables participation of mast cells in immune complex arthritis independently of Fcγ receptor modulation.

Early post-operative measurement of cytokine plasma levels combined with pre-operative bilirubin levels identify high-risk patients after liver resection.

TCR mimic monoclonal antibodies induce apoptosis of tumor cells via immune effector-independent mechanisms.

Absence of mannose-binding lectin prevents hyperglycemic cardiovascular complications.

A complement-IL-4 regulatory circuit controls liver regeneration.

Antitumor activity of a monoclonal antibody targeting major histocompatibility complex class I-Her2 peptide complexes.

Complement c5a receptor facilitates cancer metastasis by altering T-cell responses in the metastatic niche.

Complement deficiency promotes cutaneous wound healing in mice.

Pulmonary alveolar macrophages contribute to the premetastatic niche by suppressing antitumor T cell responses in the lungs.

Pyranocoumarin Tissue Distribution, Plasma Metabolome and Prostate Transcriptome Impacts of Sub-Chronic Exposure to Korean Angelica Supplement in Mice.