Silvia Brunelli

Expression of Sox3 throughout the developing central nervous system is dependent on the combined action of discrete, evolutionarily conserved regulatory elements.

Bhlhb5 is expressed in the CNS and sensory organs during mouse embryonic development.

SOX3 is required during the formation of the hypothalamo-pituitary axis.

Msx2 and necdin combined activities are required for smooth muscle differentiation in mesoangioblast stem cells.

TGFbeta/BMP activate the smooth muscle/bone differentiation programs in mesoangioblasts.

Mitochondrial biogenesis by NO yields functionally active mitochondria in mammals.

A role for MSX2 and necdin in smooth muscle differentiation of mesoangioblasts and other mesoderm progenitor cells.

Accuracy of ultrasound in the detection of liver fibrosis in chronic viral hepatitis.

Follistatin induction by nitric oxide through cyclic GMP: a tightly regulated signaling pathway that controls myoblast fusion.

Complete repair of dystrophic skeletal muscle by mesoangioblasts with enhanced migration ability.

Nitric oxide: emerging concepts about its use in cell-based therapies.

Ex vivo treatment with nitric oxide increases mesoangioblast therapeutic efficacy in muscular dystrophy.

Nitric oxide release combined with nonsteroidal antiinflammatory activity prevents muscular dystrophy pathology and enhances stem cell therapy.

Beta catenin-independent activation of MyoD in presomitic mesoderm requires PKC and depends on Pax3 transcriptional activity.

Sphingosine 1-phosphate mediates proliferation and survival of mesoangioblasts.

Necdin mediates skeletal muscle regeneration by promoting myoblast survival and differentiation.

Impaired generation of mature neurons by neural stem cells from hypomorphic Sox2 mutants.

Cell death: tipping the balance of autoimmunity and tissue repair.

Binding of sFRP-3 to EGF in the extra-cellular space affects proliferation, differentiation and morphogenetic events regulated by the two molecules.

The immune system and the repair of skeletal muscle.

Skeletal muscle differentiation of embryonic mesoangioblasts requires pax3 activity.

TGFbeta protects mesoangioblasts from apoptosis via sphingosine kinase-1 regulation.

Inflammatory and alternatively activated human macrophages attract vessel-associated stem cells, relying on separate HMGB1- and MMP-9-dependent pathways.

Necdin is expressed in cachectic skeletal muscle to protect fibers from tumor-induced wasting.

Human cardiac mesoangioblasts isolated from hypertrophic cardiomyopathies are greatly reduced in proliferation and differentiation potency.

The low-affinity receptor for neurotrophins p75NTR plays a key role for satellite cell function in muscle repair acting via RhoA.

Remodeling of the chromatin structure of the facioscapulohumeral muscular dystrophy (FSHD) locus and upregulation of FSHD-related gene 1 (FRG1) expression during human myogenic differentiation.

Biological heterogeneity of putative bladder cancer stem-like cell populations from human bladder transitional cell carcinoma samples.

Pax3:Foxc2 reciprocal repression in the somite modulates muscular versus vascular cell fate choice in multipotent progenitors.

Polarization dictates iron handling by inflammatory and alternatively activated macrophages.

Co-administration of ibuprofen and nitric oxide is an effective experimental therapy for muscular dystrophy, with immediate applicability to humans.

Skeletal muscle of gastric cancer patients expresses genes involved in muscle regeneration.

Diagnostic accuracy in coronary stenosis: comparison between visual score and quantitative analysis (quantitative computed tomographic angiography) in coronary angiography by multidetector computed tomography-coronary angiography and quantitative analysis (quantitative coronary angiography) in conventional coronary angiography.

High-mobility group box 1 release and redox regulation accompany regeneration and remodeling of skeletal muscle.

A dual acting compound releasing nitric oxide (NO) and ibuprofen, NCX 320, shows significant therapeutic effects in a mouse model of muscular dystrophy.

Sphingosine 1-phosphate induces differentiation of mesoangioblasts towards smooth muscle. A role for GATA6.

Nitric oxide sustains long-term skeletal muscle regeneration by regulating fate of satellite cells via signaling pathways requiring Vangl2 and cyclic GMP.

Nitric oxide donor and non steroidal anti inflammatory drugs as a therapy for muscular dystrophies: evidence from a safety study with pilot efficacy measures in adult dystrophic patients.

Transplanted mesoangioblasts require macrophage IL-10 for survival in a mouse model of muscle injury.

Necdin enhances myoblasts survival by facilitating the degradation of the mediator of apoptosis CCAR1/CARP1.

Cripto regulates skeletal muscle regeneration and modulates satellite cell determination by antagonizing myostatin.

Requirement of inducible nitric oxide synthase for skeletal muscle regeneration after acute damage.

Integration of 3D digital mammography with tomosynthesis for population breast-cancer screening (STORM): a prospective comparison study.

The nitric oxide-donor molsidomine modulates the innate inflammatory response in a mouse model of muscular dystrophy.

Gastric cancer does not affect the expression of atrophy-related genes in human skeletal muscle.

HuR and miR-1192 regulate myogenesis by modulating the translation of HMGB1 mRNA.

Nitric oxide drives embryonic myogenesis in chicken through the upregulation of myogenic differentiation factors.

Nitric oxide and muscle repair: multiple actions converging on therapeutic efficacy.

Incremental effect from integrating 3D-mammography (tomosynthesis) with 2D-mammography: Increased breast cancer detection evident for screening centres in a population-based trial.

Interval breast cancers: absolute and proportional incidence and blinded review in a community mammographic screening program.

Effect of integrating 3D-mammography (digital breast tomosynthesis) with 2D-mammography on radiologists' true-positive and false-positive detection in a population breast screening trial.

ADAP2 in heart development: a candidate gene for the occurrence of cardiovascular malformations in NF1 microdeletion syndrome.

Breast screening using 2D-mammography or integrating digital breast tomosynthesis (3D-mammography) for single-reading or double-reading--evidence to guide future screening strategies.

Hemogenic endothelium generates mesoangioblasts that contribute to several mesodermal lineages in vivo.

Macrophage plasticity in skeletal muscle repair.

Intracellular inactivation of thyroid hormone is a survival mechanism for muscle stem cell proliferation and lineage progression.

MeCP2 Affects Skeletal Muscle Growth and Morphology through Non Cell-Autonomous Mechanisms.

Vascular Remodelling and Mesenchymal Transition in Systemic Sclerosis.

High-throughput screening for modulators of ACVR1 transcription: discovery of potential therapeutics for fibrodysplasia ossificans progressiva.

Mesenchymal Transitions in Development and Disease.

Correction: High-throughput screening for modulators of ACVR1 transcription: discovery of potential therapeutics for fibrodysplasia ossificans progressiva.

Nitric Oxide Donor Molsidomine Positively Modulates Myogenic Differentiation of Embryonic Endothelial Progenitors.