Ariel L. Escobar

Luminal Ca2+ controls termination and refractory behavior of Ca2+-induced Ca2+ release in cardiac myocytes.

Pulsed local-field fluorescence microscopy: a new approach for measuring cellular signals in the beating heart.

Developmental changes of intracellular Ca2+ transients in beating rat hearts.

Calcium regulation of single ryanodine receptor channel gating analyzed using HMM/MCMC statistical methods.

Ryanodine receptor function in newborn rat heart.

Analysis of macroscopic ionic currents mediated by GABArho1 receptors during lanthanide modulation predicts novel states controlling channel gating.

Phospholamban phosphorylation sites enhance the recovery of intracellular Ca2+ after perfusion arrest in isolated, perfused mouse heart.

IgGs and Mabs against the beta2-adrenoreceptor block A-V conduction in mouse hearts: A possible role in the pathogenesis of ventricular arrhythmias.

Transient Ca2+ depletion of the sarcoplasmic reticulum at the onset of reperfusion.

Luminal Ca(2+) content regulates intracellular Ca(2+) release in subepicardial myocytes of intact beating mouse hearts: effect of exogenous buffers.

Beat-to-beat Ca(2+)-dependent regulation of sinoatrial nodal pacemaker cell rate and rhythm.

Calsequestrin 2 deletion shortens the refractoriness of Ca²⁺ release and reduces rate-dependent Ca²⁺-alternans in intact mouse hearts.

Intracellular Ca2+ release underlies the development of phase 2 in mouse ventricular action potentials.

Coupled gating of skeletal muscle ryanodine receptors is modulated by Ca2+, Mg2+, and ATP.

Role of inositol 1,4,5-trisphosphate in the regulation of ventricular Ca(2+) signaling in intact mouse heart.

Cardiac alternans and ventricular fibrillation: a bad case of ryanodine receptors reneging on their duty.

Ca2+ Sparks and Ca2+ waves are the subcellular events underlying Ca2+ overload during ischemia and reperfusion in perfused intact hearts.

Chasing cardiac physiology and pathology down the CaMKII cascade.

Intact Heart Loose Patch Photolysis Reveals Ionic Current Kinetics During Ventricular Action Potentials.