Sarah Derde

Insulin treatment in intensive care patients.

Changes in the central component of the hypothalamus-pituitary-thyroid axis in a rabbit model of prolonged critical illness.

Increasing intravenous glucose load in the presence of normoglycemia: effect on outcome and metabolism in critically ill rabbits.

Metabolic aspects of critical illness polyneuromyopathy.

Insufficient activation of autophagy allows cellular damage to accumulate in critically ill patients.

Thoracic epidural anaesthesia disrupts the protective mechanism of homeometric autoregulation during right ventricular pressure overload by cardiac sympathetic blockade: a randomised controlled animal study.

Muscle atrophy and preferential loss of myosin in prolonged critically ill patients.

Mitochondrial fusion, fission, and biogenesis in prolonged critically ill patients.

Critical illness induces alternative activation of M2 macrophages in adipose tissue.

Contribution of nutritional deficit to the pathogenesis of the nonthyroidal illness syndrome in critical illness: a rabbit model study.

Early parenteral nutrition evokes a phenotype of autophagy deficiency in liver and skeletal muscle of critically ill rabbits.

Increasing glucose load while maintaining normoglycemia does not evoke neuronal damage in prolonged critically ill rabbits.

Charisma: an integrated approach to automatic H&E-stained skeletal muscle cell segmentation using supervised learning and novel robust clump splitting.

Critical illness induces nutrient-independent adipogenesis and accumulation of alternatively activated tissue macrophages.

Impact of parenteral nutrition versus fasting on hepatic bile acid production and transport in a rabbit model of prolonged critical illness.

Effect of tolerating macronutrient deficit on the development of intensive-care unit acquired weakness: a subanalysis of the EPaNIC trial.

Neuropathological Correlates of Hyperglycemia During Prolonged Polymicrobial Sepsis in Mice.

Premorbid obesity, but not nutrition, prevents critical illness-induced muscle wasting and weakness.