Recherche
Enseignement
Solutions
S'identifier
FR
EN - English
CN - 中文
DE - Deutsch
ES - Español
KR - 한국어
IT - Italiano
FR - Français
PT - Português
TR - Turkish
JA - Japanese
Christian Doppler Laboratory for Biospecimen Research and Biobanking Technologies,
Institute of Pathology
Kurt Zatloukal has not added Biography.
If you are Kurt Zatloukal and would like to personalize this page please email our Author Liaison for assistance.
Endometrial stromal sarcomas frequently express epidermal growth factor receptor (EGFR, HER-1): potential basis for a new therapeutic approach.
The American journal of surgical pathology Apr, 2005 | Pubmed ID: 15767802
CAR and PXR agonists stimulate hepatic bile acid and bilirubin detoxification and elimination pathways in mice.
Hepatology (Baltimore, Md.) Aug, 2005 | Pubmed ID: 15986414
Role of nuclear receptors and hepatocyte-enriched transcription factors for Ntcp repression in biliary obstruction in mouse liver.
American journal of physiology. Gastrointestinal and liver physiology Nov, 2005 | Pubmed ID: 16002565
Lithocholic acid feeding induces segmental bile duct obstruction and destructive cholangitis in mice.
The American journal of pathology Feb, 2006 | Pubmed ID: 16436656
24-norUrsodeoxycholic acid is superior to ursodeoxycholic acid in the treatment of sclerosing cholangitis in Mdr2 (Abcb4) knockout mice.
Gastroenterology Feb, 2006 | Pubmed ID: 16472600
Hepatobiliary transporter expression in intercellular adhesion molecule 1 knockout and Fas receptor-deficient mice after common bile duct ligation is independent of the degree of inflammation and oxidative stress.
Drug metabolism and disposition: the biological fate of chemicals Sep, 2007 | Pubmed ID: 17576807
A new xenobiotic-induced mouse model of sclerosing cholangitis and biliary fibrosis.
The American journal of pathology Aug, 2007 | Pubmed ID: 17600122
Side chain structure determines unique physiologic and therapeutic properties of norursodeoxycholic acid in Mdr2-/- mice.
Hepatology (Baltimore, Md.) Jun, 2009 | Pubmed ID: 19475687
Caleydo: connecting pathways and gene expression.
Bioinformatics (Oxford, England) Oct, 2009 | Pubmed ID: 19620095
Farnesoid X receptor critically determines the fibrotic response in mice but is expressed to a low extent in human hepatic stellate cells and periductal myofibroblasts.
The American journal of pathology Dec, 2009 | Pubmed ID: 19910507
Sensitivity and specificity of in situ proximity ligation for protein interaction analysis in a model of steatohepatitis with Mallory-Denk bodies.
PloS one , 2014 | Pubmed ID: 24798445
The impact of free or standardized lifestyle and urine sampling protocol on metabolome recognition accuracy.
Genes & nutrition Jan, 2015 | Pubmed ID: 25403096
Immunofluorescence and Immunohistochemical Detection of Keratins.
Methods in enzymology , 2016 | Pubmed ID: 26795470
Pathogen Inactivating Properties and Increased Sensitivity in Molecular Diagnostics by PAXgene, a Novel Non-Crosslinking Tissue Fixative.
PloS one , 2016 | Pubmed ID: 26974150
Individual Human Metabolic Phenotype Analyzed by (1)H NMR of Saliva Samples.
Journal of proteome research Jun, 2016 | Pubmed ID: 27087681
Protocol for HER2 FISH determination on PAXgene-fixed and paraffin-embedded tissue in breast cancer.
International journal of experimental pathology 04, 2016 | Pubmed ID: 27273709
Medical University Graz
Martina Loibner1,2,
Lisa Oberauner-Wappis1,2,
Christian Viertler2,
Daniel Groelz3,
Kurt Zatloukal1,2
1Christian Doppler Laboratory for Biospecimen Research and Biobanking Technologies, Institute of Pathology, Medical University Graz,
2Institute of Pathology, Medical University Graz,
3Research and Development, Qiagen GmbH
Confidentialité
Conditions d'utilisation
Politiques
Contactez-nous
RECOMMANDER À LA BIBLIOTHÈQUE
NEWSLETTERS JoVE
JoVE Journal
Collections de méthodes
JoVE Encyclopedia of Experiments
Archives
JoVE Core
JoVE Business
JoVE Science Education
JoVE Lab Manual
Centre de ressources universitaires
Auteurs
Bibliothécaires
Accès
À PROPOS DE JoVE
Copyright © 2024 MyJoVE Corporation. Tous droits réservés.