Seung Keun Back

A mouse model for peripheral neuropathy produced by a partial injury of the nerve supplying the tail.

Increases in spinal vasoactive intestinal polypeptide and neuropeptide Y are not sufficient for the genesis of neuropathic pain in rats.

Ascending pathways for mechanical allodynia in a rat model of neuropathic pain.

Intraarticular pretreatment with ketamine and memantine could prevent arthritic pain: relevance to the decrease of spinal c-fos expression in rats.

Gabapentin relieves mechanical, warm and cold allodynia in a rat model of peripheral neuropathy.

Dorsal column lesion reduces mechanical allodynia in the induction, but not the maintenance, phase in spinal hemisected rats.

Loss of spinal mu-opioid receptor is associated with mechanical allodynia in a rat model of peripheral neuropathy.

Induction of total insensitivity to capsaicin and hypersensitivity to garlic extract in human by decreased expression of TRPV1.

Developmental characteristics of neuropathic pain induced by peripheral nerve injury of rats during neonatal period.

Impairment of long-term depression induced by chronic brain inflammation in rats.

Long-lasting neonatal inflammation enhances pain responses to subsequent inflammation, but not peripheral nerve injury in adult rats.

Membrane-delimited coupling of TRPV1 and mGluR5 on presynaptic terminals of nociceptive neurons.

Complete Freund's adjuvant-induced intervertebral discitis as an animal model for discogenic low back pain.

Synthesis and bradykinin inhibitory activity of novel non-peptide compounds, and evaluation of in vivo analgesic activity.

Are spinal GABAergic elements related to the manifestation of neuropathic pain in rat?

Synthesis and T-type calcium channel blocking activity of novel diphenylpiperazine compounds, and evaluation of in vivo analgesic activity.

A novel rat forelimb model of neuropathic pain produced by partial injury of the median and ulnar nerves.

Analgesic effect of highly reversible ω-conotoxin FVIA on N type Ca2+ channels.

TRPV1 in GABAergic interneurons mediates neuropathic mechanical allodynia and disinhibition of the nociceptive circuitry in the spinal cord.

The calcium-activated chloride channel anoctamin 1 acts as a heat sensor in nociceptive neurons.

Chronically relapsing pruritic dermatitis in the rats treated as neonate with capsaicin; a potential rat model of human atopic dermatitis.

HYP-1, a novel diamide compound, relieves inflammatory and neuropathic pain in rats.

Juvenile obesity aggravates disease severity in a rat model of atopic dermatitis.

Brief Isolation Changes Nociceptive Behaviors and Compromises Drug Tests in Mice.

Formaldehyde-Induced Aggravation of Pruritus and Dermatitis Is Associated with the Elevated Expression of Th1 Cytokines in a Rat Model of Atopic Dermatitis.

Acute inflammation reveals GABAA receptor-mediated nociception in mouse dorsal root ganglion neurons via PGE2 receptor 4 signaling.

Asthma-like airway inflammation and responses in a rat model of atopic dermatitis induced by neonatal capsaicin treatment.