Daniel F. Jarosz

A single amino acid governs enhanced activity of DinB DNA polymerases on damaged templates.

Y-family DNA polymerases respond to DNA damage-independent inhibition of replication fork progression.

Characterization of Escherichia coli translesion synthesis polymerases and their accessory factors.

Y-family DNA polymerases in Escherichia coli.

DNA polymerase V allows bypass of toxic guanine oxidation products in vivo.

Proficient and accurate bypass of persistent DNA lesions by DinB DNA polymerases.

UmuD and RecA directly modulate the mutagenic potential of the Y family DNA polymerase DinB.

A DinB variant reveals diverse physiological consequences of incomplete TLS extension by a Y-family DNA polymerase.

HSP90 at the hub of protein homeostasis: emerging mechanistic insights.

Protein homeostasis and the phenotypic manifestation of genetic diversity: principles and mechanisms.

Hsp90 and environmental stress transform the adaptive value of natural genetic variation.

Song: SOS (To the Tune of ABBA's "SOS").

Prions are a common mechanism for phenotypic inheritance in wild yeasts.

Pernicious pathogens or expedient elements of inheritance: the significance of yeast prions.

An evolutionarily conserved prion-like element converts wild fungi from metabolic specialists to generalists.

Cross-kingdom chemical communication drives a heritable, mutually beneficial prion-based transformation of metabolism.

Rebels with a cause: molecular features and physiological consequences of yeast prions.

Intrinsically Disordered Proteins Drive Emergence and Inheritance of Biological Traits.

A common bacterial metabolite elicits prion-based bypass of glucose repression.

Amyloid Prions in Fungi.

Old moms say, no Sir.

Comprehensive and quantitative mapping of RNA-protein interactions across a transcribed eukaryotic genome.