Anita Ignatius

[Possibilities and limits in tissue engineering of the anterior cruciate ligament].

The anaphylatoxin receptor C5aR is present during fracture healing in rats and mediates osteoblast migration in vitro.

Complement C3a and C5a modulate osteoclast formation and inflammatory response of osteoblasts in synergism with IL-1β.

The impact of low-magnitude high-frequency vibration on fracture healing is profoundly influenced by the oestrogen status in mice.

In vivo performance of novel soybean/gelatin-based bioactive and injectable hydroxyapatite foams.

Midkine-deficiency delays chondrogenesis during the early phase of fracture healing in mice.

Small changes in bone structure of female α7 nicotinic acetylcholine receptor knockout mice.

Mechanical stimulation of human tendon stem/progenitor cells results in upregulation of matrix proteins, integrins and MMPs, and activation of p38 and ERK1/2 kinases.

Impaired extracellular matrix structure resulting from malnutrition in ovariectomized mature rats.

The molecular fingerprint of lung inflammation after blunt chest trauma.

Hypochlorhydria-induced calcium malabsorption does not affect fracture healing but increases post-traumatic bone loss in the intact skeleton.

Antagonizing midkine accelerates fracture healing in mice by enhanced bone formation in the fracture callus.

Inhibition of Midkine Augments Osteoporotic Fracture Healing.

Evaluation of high-resolution In Vivo MRI for longitudinal analysis of endochondral fracture healing in mice.

Calcium and vitamin-D deficiency marginally impairs fracture healing but aggravates posttraumatic bone loss in osteoporotic mice.

Chronic psychosocial stress disturbs long-bone growth in adolescent mice.

Complement receptors C5aR1 and C5aR2 act differentially during the early immune response after bone fracture but are similarly involved in bone repair.