Charles Steenbergen

Glycogen synthase kinase 3 inhibition slows mitochondrial adenine nucleotide transport and regulates voltage-dependent anion channel phosphorylation.

Cardioprotection and altered mitochondrial adenine nucleotide transport.

Estrogen receptor-beta activation results in S-nitrosylation of proteins involved in cardioprotection.

Autologous mesenchymal stem cells produce reverse remodelling in chronic ischaemic cardiomyopathy.

Engraftment, differentiation, and functional benefits of autologous cardiosphere-derived cells in porcine ischemic cardiomyopathy.

Adenoviral transfer of HIF-1alpha enhances vascular responses to critical limb ischemia in diabetic mice.

Sex differences in the phosphorylation of mitochondrial proteins result in reduced production of reactive oxygen species and cardioprotection in females.

VAMP-1, VAMP-2, and syntaxin-4 regulate ANP release from cardiac myocytes.

Functional neovascularization of biodegradable dextran hydrogels with multiple angiogenic growth factors.

Mechanism of cardioprotection: what can we learn from females?

Ischemic preconditioning attenuates mitochondrial localization of PTEN induced by ischemia-reperfusion.

Controlled activation of morphogenesis to generate a functional human microvasculature in a synthetic matrix.

Impact of scar, viable myocardium, and epicardial fat on substrate identification of ventricular tachycardia in a case with nonischemic cardiomyopathy.

Cysteine 203 of cyclophilin D is critical for cyclophilin D activation of the mitochondrial permeability transition pore.

Mitochondrial adenine nucleotide transport and cardioprotection.

Mitochondrial permeability transition pore and calcium handling.

Dextran hydrogel scaffolds enhance angiogenic responses and promote complete skin regeneration during burn wound healing.

Creatine kinase-mediated improvement of function in failing mouse hearts provides causal evidence the failing heart is energy starved.

Endothelial expression of hypoxia-inducible factor 1 protects the murine heart and aorta from pressure overload by suppression of TGF-β signaling.

Nuclear miRNA regulates the mitochondrial genome in the heart.

A composite polymer nanoparticle overcomes multidrug resistance and ameliorates doxorubicin-associated cardiomyopathy.

Measurement of S-nitrosylation occupancy in the myocardium with cysteine-reactive tandem mass tags: short communication.

Creatine kinase overexpression improves ATP kinetics and contractile function in postischemic myocardium.

Integration and regression of implanted engineered human vascular networks during deep wound healing.

Skeletal muscle ATP kinetics are impaired in frail mice.

Cardioprotection and myocardial reperfusion: pitfalls to clinical application.

Creatine kinase-overexpression improves myocardial energetics, contractile dysfunction and survival in murine doxorubicin cardiotoxicity.

Hydrogen sulfide [corrected] increases survival during sepsis: protective effect of CHOP inhibition.

Strengthening the skin with topical delivery of keratinocyte growth factor-1 using a novel DNA plasmid.

S-nitrosylation of TRIM72 at cysteine 144 is critical for protection against oxidation-induced protein degradation and cell death.

Pivotal role of mTORC2 and involvement of ribosomal protein S6 in cardioprotective signaling.

miR-181c regulates the mitochondrial genome, bioenergetics, and propensity for heart failure in vivo.

Glyceraldehyde-3-phosphate dehydrogenase acts as a mitochondrial trans-S-nitrosylase in the heart.

The NHLBI-sponsored Consortium for preclinicAl assESsment of cARdioprotective therapies (CAESAR): a new paradigm for rigorous, accurate, and reproducible evaluation of putative infarct-sparing interventions in mice, rabbits, and pigs.

ErbB2 overexpression upregulates antioxidant enzymes, reduces basal levels of reactive oxygen species, and protects against doxorubicin cardiotoxicity.

Hyaluronic acid-serum hydrogels rapidly restore metabolism of encapsulated stem cells and promote engraftment.

Characterization of the sex-dependent myocardial S-nitrosothiol proteome.

Exosomal MicroRNA-15a Transfer from the Pancreas Augments Diabetic Complications by Inducing Oxidative Stress.

Divergent Effects of miR-181 Family Members on Myocardial Function Through Protective Cytosolic and Detrimental Mitochondrial microRNA Targets.

miR-181b regulates vascular stiffness age dependently in part by regulating TGF-β signaling.

Adenosine A1 receptor activation increases myocardial protein S-nitrosothiols and elicits protection from ischemia-reperfusion injury in male and female hearts.

Genetic testing improves identification of transthyretin amyloid (ATTR) subtype in cardiac amyloidosis.