Laurie E. Kilpatrick

Regulation of TNF-induced oxygen radical production in human neutrophils: role of delta-PKC.

Protection against sepsis-induced lung injury by selective inhibition of protein kinase C-δ (δ-PKC).

Prasugrel metabolites inhibit neutrophil functions.

Protein kinase C and acute respiratory distress syndrome.

What's new in Shock? July 2013.

LPS-induced systemic inflammation is more severe in P2Y12 null mice.

Pulmonary endothelial protein kinase C-delta (PKCδ) regulates neutrophil migration in acute lung inflammation.

CGX1037 is a novel PKC isoform delta selective inhibitor in platelets.

The role of P2Y₁₂ receptor and activated platelets during inflammation.

Biodistribution and Efficacy of Targeted Pulmonary Delivery of a Protein Kinase C-δ Inhibitory Peptide: Impact on Indirect Lung Injury.

P2Y12 Receptor Modulates Sepsis-Induced Inflammation.

A novel microfluidic assay reveals a key role for protein kinase C δ in regulating human neutrophil-endothelium interaction.

Role of Protein Kinase C-delta in regulating platelet activation and platelet-leukocyte interaction during sepsis.

PKCδ inhibition as a novel medical countermeasure for radiation-induced vascular damage.

Protein Kinase C-Delta (PKCδ) Tyrosine Phosphorylation is a Critical Regulator of Neutrophil-Endothelial Cell Interaction in Inflammation.

Protein kinase C-delta inhibition protects blood-brain barrier from sepsis-induced vascular damage.

The Role of Tyrosine Phosphorylation of Protein Kinase C Delta in Infection and Inflammation.

Protein kinase C-delta inhibition is organ-protective, enhances pathogen clearance, and improves survival in sepsis.

Neutrophil-endothelial interactions of murine cells is not a good predictor of their interactions in human cells.

Experimental Approaches to Evaluate Leukocyte-Endothelial Cell Interactions in Sepsis and Inflammation.

Mechanisms of radiation-induced endothelium damage: Emerging models and technologies.

Transient Induction of Fever in the Imiquimod C57BL/6 Mouse Model of Psoriasis-Like Disease Involves IL-1 and IL-6 but Not IL-36.

Emerging Approaches to Understanding Microvascular Endothelial Heterogeneity: A Roadmap for Developing Anti-Inflammatory Therapeutics.