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The Heart Institute,
Division of Molecular Cardiovascular Biology,
The Heart Institute, Division of Molecular Cardiovascular Biology
Catherine A. Makarewich has not added Biography.
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Ca(2+) influx through L-type Ca(2+) channels and transient receptor potential channels activates pathological hypertrophy signaling.
Journal of molecular and cellular cardiology Nov, 2012 | Pubmed ID: 22921230
Transient receptor potential channels contribute to pathological structural and functional remodeling after myocardial infarction.
Circulation research Aug, 2014 | Pubmed ID: 25047165
STIM1 elevation in the heart results in aberrant Ca²⁺ handling and cardiomyopathy.
Journal of molecular and cellular cardiology Oct, 2015 | Pubmed ID: 26241845
A Tension-Based Model Distinguishes Hypertrophic versus Dilated Cardiomyopathy.
Cell May, 2016 | Pubmed ID: 27114035
Caveolae-localized L-type Ca2+ channels do not contribute to function or hypertrophic signalling in the mouse heart.
Cardiovascular research Jun, 2017 | Pubmed ID: 28402392
The hidden world of membrane microproteins.
Experimental cell research 03, 2020 | Pubmed ID: 31978386
Gene Therapy With the DWORF Micropeptide Attenuates Cardiomyopathy in Mice.
Circulation research 10, 2020 | Pubmed ID: 32878549
The cardiac-enriched microprotein mitolamban regulates mitochondrial respiratory complex assembly and function in mice.
Proceedings of the National Academy of Sciences of the United States of America 02, 2022 | Pubmed ID: 35101990
Exercise-Induced Long Noncoding RNAs As New Players in Cardiac Hypertrophy.
Circulation 04, 2022 | Pubmed ID: 35436132
Cincinnati Children's Hospital Medical Center
University of Cincinnati College of Medicine
Omar Brito-Estrada*,1,
Keira R. Hassel*,1,
Catherine A. Makarewich1,2
1The Heart Institute, Division of Molecular Cardiovascular Biology, Cincinnati Children's Hospital Medical Center,
2Department of Pediatrics, University of Cincinnati College of Medicine
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