Waliul I. Khan

Visceral hyperalgesia and intestinal dysmotility in a mouse model of postinfective gut dysfunction.

CD4+ T-cell modulation of visceral nociception in mice.

Antidepressants attenuate increased susceptibility to colitis in a murine model of depression.

IL-9 enhances growth of ICC, maintains network structure and strengthens rhythmicity of contraction in culture.

CD4+ T cell-mediated immunological control of enterochromaffin cell hyperplasia and 5-hydroxytryptamine production in enteric infection.

Cytokine modulation of muscarinic receptors in the murine intestine.

Reactivation of inflammatory bowel disease in a mouse model of depression.

CD4+ T-cell-mediated anti-tumor immunity can be uncoupled from autoimmunity via the STAT4/STAT6 signaling axis.

Serotonin has a key role in pathogenesis of experimental colitis.

Mucin gene deficiency in mice impairs host resistance to an enteric parasitic infection.

Chronic gastrointestinal inflammation induces anxiety-like behavior and alters central nervous system biochemistry in mice.

Mechanisms underlying gut dysfunction in a murine model of chronic parasitic infection.

A mouse model of airway disease: oncostatin M-induced pulmonary eosinophilia, goblet cell hyperplasia, and airway hyperresponsiveness are STAT6 dependent, and interstitial pulmonary fibrosis is STAT6 independent.

Adoptive transfer of macrophage from mice with depression-like behavior enhances susceptibility to colitis.

Serotonin activates dendritic cell function in the context of gut inflammation.