R. Jeroen Pasterkamp

MICALs, a family of conserved flavoprotein oxidoreductases, function in plexin-mediated axonal repulsion.

Semaphorin junction: making tracks toward neural connectivity.

Semaphorin 7A promotes axon outgrowth through integrins and MAPKs.

R-Ras fills another GAP in semaphorin signalling.

Soluble CD100 functions on human monocytes and immature dendritic cells require plexin C1 and plexin B1, respectively.

MICAL flavoprotein monooxygenases: expression during neural development and following spinal cord injuries in the rat.

High-resolution structure of the catalytic region of MICAL (molecule interacting with CasL), a multidomain flavoenzyme-signaling molecule.

Semaphorins in axon regeneration: developmental guidance molecules gone wrong?

Semaphorin 7A initiates T-cell-mediated inflammatory responses through alpha1beta1 integrin.

MICAL flavoprotein monooxygenases: structure, function and role in semaphorin signaling.

Expression patterns of semaphorin7A and plexinC1 during rat neural development suggest roles in axon guidance and neuronal migration.

Getting connected in the dopamine system.

Semaphorin signaling: progress made and promises ahead.

Axon guidance proteins: novel therapeutic targets for ALS?

Semaphorin function in neural plasticity and disease.

MeCP2 deficiency disrupts axonal guidance, fasciculation, and targeting by altering Semaphorin 3F function.

Development and engineering of dopamine neurons. Preface.

Axon guidance in the dopamine system.

Genome-wide association study identifies 19p13.3 (UNC13A) and 9p21.2 as susceptibility loci for sporadic amyotrophic lateral sclerosis.

Semaphorin 3F is a bifunctional guidance cue for dopaminergic axons and controls their fasciculation, channeling, rostral growth, and intracortical targeting.

FUS mutations in familial amyotrophic lateral sclerosis in the Netherlands.

Semaphorin signaling: molecular switches at the midline.

Neuropeptide delivery to the brain: a von Willebrand factor signal peptide to direct neuropeptide secretion.

Wnt/planar cell polarity signaling controls the anterior-posterior organization of monoaminergic axons in the brainstem.

Rab6, Rab8, and MICAL3 cooperate in controlling docking and fusion of exocytotic carriers.

MICAL-1 is a negative regulator of MST-NDR kinase signaling and apoptosis.

MICALs in control of the cytoskeleton, exocytosis, and cell death.

Kinetic and spectroscopic characterization of the putative monooxygenase domain of human MICAL-1.

VCP mutations in familial and sporadic amyotrophic lateral sclerosis.

UNC13A is a modifier of survival in amyotrophic lateral sclerosis.

Angiogenin variants in Parkinson disease and amyotrophic lateral sclerosis.

NIPA1 polyalanine repeat expansions are associated with amyotrophic lateral sclerosis.