S'identifier

Elias A. Rahal

Department of Experimental Pathology,
Immunology and Microbiology,
Faculty of Medicine,
Center for Infectious Diseases Research (CIDR),
Department of Experimental Pathology, Immunology and Microbiology, Faculty of Medicine,
Center for Infectious Diseases Research (CIDR), Faculty of Medicine

American University of Beirut

BIOGRAPHY

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PUBLICATIONS

Role of rifampicin in limiting Escherichia coli O157:H7 Shiga-like toxin expression and enhancement of survival of infected BALB/c mice.

International journal of antimicrobial agents Feb, 2011 | Pubmed ID: 21129928

Decrease in Shiga toxin expression using a minimal inhibitory concentration of rifampicin followed by bactericidal gentamicin treatment enhances survival of Escherichia coli O157:H7-infected BALB/c mice.

Annals of clinical microbiology and antimicrobials Sep, 2011 | Pubmed ID: 21906403

The impact of prophylactic antiviral agents and statin administration on graft longevity in kidney allograft recipients.

Immunopharmacology and immunotoxicology Oct, 2012 | Pubmed ID: 22292901

Escherichia coli O157:H7-Clinical aspects and novel treatment approaches.

Frontiers in cellular and infection microbiology , 2012 | Pubmed ID: 23162800

Effects of subinhibitory concentrations of antimicrobial agents on Escherichia coli O157:H7 Shiga toxin release and role of the SOS response.

Foodborne pathogens and disease Sep, 2013 | Pubmed ID: 23808851

Effect of rifampicin and gentamicin on Shiga toxin 2 expression level and the SOS response in Escherichia coli O104:H4.

Foodborne pathogens and disease Jan, 2015 | Pubmed ID: 25383748

Approaches to treatment of emerging Shiga toxin-producing Escherichia coli infections highlighting the O104:H4 serotype.

Frontiers in cellular and infection microbiology , 2015 | Pubmed ID: 25853096

Epstein-Barr Virus and Human herpes virus 6 Type A DNA Enhance IL-17 Production in Mice.

Viral immunology Jun, 2015 | Pubmed ID: 25870901

Atorvastatin Reduces the Survival of Candida albicans-Infected BALB/c Mice.

Frontiers in microbiology , 2015 | Pubmed ID: 26732740

Epstein-Barr Virus DNA Enhances Diptericin Expression and Increases Hemocyte Numbers in via the Immune Deficiency Pathway.

Frontiers in microbiology , 2018 | Pubmed ID: 29942298

Epstein-Barr virus DNA modulates regulatory T-cell programming in addition to enhancing interleukin-17A production via Toll-like receptor 9.

PloS one , 2018 | Pubmed ID: 29995930

The role of viral infections in the development of autoimmune diseases.

Critical reviews in microbiology Aug, 2019 | Pubmed ID: 31145640

Endosomal Toll-Like Receptors Mediate Enhancement of Interleukin-17A Production Triggered by Epstein-Barr Virus DNA in Mice.

Journal of virology 10, 2019 | Pubmed ID: 31375581

Atorvastatin increases the production of proinflammatory cytokines and decreases the survival of Escherichia coli-infected mice.

Scientific reports 08, 2019 | Pubmed ID: 31406240

Endosomal Toll-Like Receptors (TLRs) mediate enhancement of IL-17A production triggered by Epstein-Barr virus (EBV) DNA in mice.

Journal of infection in developing countries Feb, 2018 | Pubmed ID: 31805001

Triple Immunotherapy Overcomes Immune Evasion by Tumor in a Melanoma Mouse Model.

Frontiers in oncology , 2020 | Pubmed ID: 32596146

as a Model System to Assess the Effect of Epstein-Barr Virus DNA on Inflammatory Gut Diseases.

Frontiers in immunology , 2021 | Pubmed ID: 33828545

Effect of Epstein-Barr Virus DNA on the Incidence and Severity of Arthritis in a Rheumatoid Arthritis Mouse Model.

Frontiers in immunology , 2021 | Pubmed ID: 34040613

Epstein-Barr Virus DNA Exacerbates Colitis Symptoms in a Mouse Model of Inflammatory Bowel Disease.

Viruses 06, 2021 | Pubmed ID: 34210024

IL-17A in COVID-19 Cases: a meta-analysis.

Journal of infection in developing countries 11, 2021 | Pubmed ID: 34898490

Methods for fighting emerging pathogens.

Nature methods 04, 2022 | Pubmed ID: 35396474

Editorial: The microbial modulation of autoimmune processes and proinflammatory pathways.

Frontiers in cellular and infection microbiology , 2022 | Pubmed ID: 35982782

INSTITUTIONS

American University of Beirut

JOVE ARTICLES

Isolation and Quantification of Epstein-Barr Virus from the P3HR1 Cell Line

Elio R. Bitar¹,

Marcel S. Shams Eddin¹,

Elias A. Rahal^1,2

¹Department of Experimental Pathology, Immunology and Microbiology, Faculty of Medicine, American University of Beirut,

²Center for Infectious Diseases Research (CIDR), Faculty of Medicine, American University of Beirut

Immunology and Infection

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Elias A. Rahal

Department of Experimental Pathology,Immunology and Microbiology,Faculty of Medicine,Center for Infectious Diseases Research (CIDR),Department of Experimental Pathology, Immunology and Microbiology, Faculty of Medicine,Center for Infectious Diseases Research (CIDR), Faculty of Medicine

American University of Beirut

Isolation and Quantification of Epstein-Barr Virus from the P3HR1 Cell Line

Department of Experimental Pathology,
Immunology and Microbiology,
Faculty of Medicine,
Center for Infectious Diseases Research (CIDR),
Department of Experimental Pathology, Immunology and Microbiology, Faculty of Medicine,
Center for Infectious Diseases Research (CIDR), Faculty of Medicine