Yuemei Dong

Anopheles infection responses; laboratory models versus field malaria transmission systems.

AgDscam, a hypervariable immunoglobulin domain-containing receptor of the Anopheles gambiae innate immune system.

Anopheles gambiae immune responses to human and rodent Plasmodium parasite species.

Spatial and sex-specific dissection of the Anopheles gambiae midgut transcriptome.

Evolutionary dynamics of immune-related genes and pathways in disease-vector mosquitoes.

Continuous exposure to Plasmodium results in decreased susceptibility and transcriptomic divergence of the Anopheles gambiae immune system.

Anopheles fibrinogen-related proteins provide expanded pattern recognition capacity against bacteria and malaria parasites.

Implication of the mosquito midgut microbiota in the defense against malaria parasites.

Pathogenomics of Culex quinquefasciatus and meta-analysis of infection responses to diverse pathogens.

Natural microbe-mediated refractoriness to Plasmodium infection in Anopheles gambiae.

The entomopathogenic fungus Beauveria bassiana activate toll and JAK-STAT pathway-controlled effector genes and anti-dengue activity in Aedes aegypti.

Engineered anopheles immunity to Plasmodium infection.

Anopheles Imd pathway factors and effectors in infection intensity-dependent anti-Plasmodium action.

Transcriptional mediators Kto and Skd are involved in the regulation of the IMD pathway and anti-Plasmodium defense in Anopheles gambiae.

Anopheles NF-κB-regulated splicing factors direct pathogen-specific repertoires of the hypervariable pattern recognition receptor AgDscam.

Caudal is a negative regulator of the Anopheles IMD pathway that controls resistance to Plasmodium falciparum infection.

The Anopheles innate immune system in the defense against malaria infection.

Impact of trehalose transporter knockdown on Anopheles gambiae stress adaptation and susceptibility to Plasmodium falciparum infection.

Exploring Anopheles gut bacteria for Plasmodium blocking activity.

Chromobacterium Csp_P reduces malaria and dengue infection in vector mosquitoes and has entomopathogenic and in vitro anti-pathogen activities.

Cytoplasmic actin is an extracellular insect immune factor which is secreted upon immune challenge and mediates phagocytosis and direct killing of bacteria, and is a Plasmodium Antagonist.

Anopheles Midgut FREP1 Mediates Plasmodium Invasion.

The Anopheles FBN9 immune factor mediates Plasmodium species-specific defense through transgenic fat body expression.

Changes in the microbiota cause genetically modified to spread in a population.

Immune Regulation of Is Species Specific and Infection Intensity Dependent.

CRISPR/Cas9 -mediated gene knockout of Anopheles gambiae FREP1 suppresses malaria parasite infection.

The mosquito adulticidal Chromobacterium sp. Panama causes transgenerational impacts on fitness parameters and elicits xenobiotic gene responses.

Aminopeptidase secreted by Chromobacterium sp. Panama inhibits dengue virus infection by degrading the E protein.

Lacking Inefficiently Transmits to Mice.

Disruption of mosGILT in Anopheles gambiae impairs ovarian development and Plasmodium infection.

Broad spectrum immunomodulatory effects of Anopheles gambiae microRNAs and their use for transgenic suppression of Plasmodium.

Versatile transgenic multistage effector-gene combinations for suppression in .

Prospects and Pitfalls: Next-Generation Tools to Control Mosquito-Transmitted Disease.

Field-deployable molecular diagnostic platform for arbovirus detection in Aedes aegypti.

Glyphosate inhibits melanization and increases susceptibility to infection in insects.

Mosquito transgenesis for malaria control.

Transcriptome profiles of Anopheles gambiae harboring natural low-level Plasmodium infection reveal adaptive advantages for the mosquito.

C-type lectin 4 regulates broad-spectrum melanization-based refractoriness to malaria parasites.

The Aedes aegypti siRNA pathway mediates broad-spectrum defense against human pathogenic viruses and modulates antibacterial and antifungal defenses.