Joseph M. Wallace

Applications of atomic force microscopy for the assessment of nanoscale morphological and mechanical properties of bone.

Nanoscale structure of type I collagen fibrils: quantitative measurement of D-spacing.

Elevated mechanical loading when young provides lifelong benefits to cortical bone properties in female rats independent of a surgically induced menopause.

Nanoscale changes in collagen are reflected in physical and mechanical properties of bone at the microscale in diabetic rats.

Multiscale analysis of morphology and mechanics in tail tendon from the ZDSD rat model of type 2 diabetes.

Physical weight loading induces expression of tryptophan hydroxylase 2 in the brain stem.

Comment on "The ultrastructure of type I collagen at nanoscale: large or small D-spacing distribution?" by H.-N. Su, L.-Y. Ran, Z.-H. Chen, Q.-L. Qin, M. Shi, X.-Y. Song, X.-L. Chen, Y.-Z. Zhang and B.-B. Xie, Nanoscale, 2014, 6, 8134.

Differential effects of Epigallocatechin-3-gallate containing supplements on correcting skeletal defects in a Down syndrome mouse model.

Removing or truncating connexin 43 in murine osteocytes alters cortical geometry, nanoscale morphology, and tissue mechanics in the tibia.

Treadmill Exercise Improves Fracture Toughness and Indentation Modulus without Altering the Nanoscale Morphology of Collagen in Mice.

Ligand trap of the activin receptor type IIA inhibits osteoclast stimulation of bone remodeling in diabetic mice with chronic kidney disease.

Salubrinal improves mechanical properties of the femur in osteogenesis imperfecta mice.

Even with rehydration, preservation in ethanol influences the mechanical properties of bone and how bone responds to experimental manipulation.

Connexin37 deficiency alters organic bone matrix, cortical bone geometry, and increases Wnt/β-catenin signaling.

Corrigendum to "Even with rehydration, preservation in ethanol influences the mechanical properties of bone and how bone responds to experimental manipulation" [Bone 97 (2017) 49-53].

Zoledronate and Raloxifene combination therapy enhances material and mechanical properties of diseased mouse bone.

Stat3 in osteocytes mediates osteogenic response to loading.

Substrate Strain Mitigates Effects of β-Aminopropionitrile-Induced Reduction in Enzymatic Crosslinking.

Muscle contraction induces osteogenic levels of cortical bone strain despite muscle weakness in a mouse model of Osteogenesis Imperfecta.

The loss of STAT3 in mature osteoclasts has detrimental effects on bone structure.

Limited impacts of thermoneutral housing on bone morphology and mechanical properties in growing female mice exposed to external loading and raloxifene treatment.

MicroRNAs and osteocytes.

Nmp4, a Regulator of Induced Osteoanabolism, Also Influences Insulin Secretion and Sensitivity.

Skeletal Deficits in Male and Female down Syndrome Model Mice Arise Independent of Normalized Dyrk1a Expression in Osteoblasts.

Increased dosage and treatment time of Epigallocatechin-3-gallate (EGCG) negatively affects skeletal parameters in normal mice and Down syndrome mouse models.

Dietary supplements do not improve bone morphology or mechanical properties in young female C57BL/6 mice.

Raloxifene Stimulates Estrogen Signaling to Protect Against Age- and Sex-Related Intervertebral Disc Degeneration in Mice.

A novel murine model of combined insulin-dependent diabetes and chronic kidney disease has greater skeletal detriments than either disease individually.

Combined Thermoneutral Housing and Raloxifene Treatment Improves Trabecular Bone Microarchitecture and Strength in Growing Female Mice.

Genetic dissection of triplicated chromosome 21 orthologs yields varying skeletal traits in Down syndrome model mice.

Correlation analysis of cartilage wear with biochemical composition, viscoelastic properties and friction.

Fragile X Messenger Ribonucleoprotein 1 (FMR1), a novel inhibitor of osteoblast/osteocyte differentiation, regulates bone formation, mass, and strength in young and aged male and female mice.

Ex vivo exposure to calcitonin or raloxifene improves mechanical properties of diseased bone through non-cell mediated mechanisms.