Justin Parreno

Redifferentiated Chondrocytes in Fibrin Gel for the Repair of Articular Cartilage Lesions.

Age-related changes in eye lens biomechanics, morphology, refractive index and transparency.

Tropomyosin 3.1 Association With Actin Stress Fibers is Required for Lens Epithelial to Mesenchymal Transition.

Whole mount staining of lenses for visualization of lens epithelial cell proteins.

Stress deprivation of tendon explants or Tpm3.1 inhibition in tendon cells reduces F-actin to promote a tendinosis-like phenotype.

Methodologies to unlock the molecular expression and cellular structure of ocular lens epithelial cells.

Optics-Free, In Situ Swelling Monitoring of Articular Cartilage with Graphene Strain Sensors.

Nonmuscle Myosin IIA Regulates the Precise Alignment of Hexagonal Eye Lens Epithelial Cells During Fiber Cell Formation and Differentiation.

Overload in a Rat In Vivo Model of Synergist Ablation Induces Tendon Multiscale Structural and Functional Degeneration.

The application of mechanical load onto mouse tendons by magnetic restraining represses Mmp-3 expression.

Adseverin, an actin-binding protein, modulates hypertrophic chondrocyte differentiation and osteoarthritis progression.

Targeting F-actin stress fibers to suppress the dedifferentiated phenotype in chondrocytes.

The Actin Cytoskeleton as a Regulator of Proteoglycan 4.