Tomas Falzone

Authentic cell-specific and developmentally regulated expression of pro-opiomelanocortin genomic fragments in hypothalamic and hindbrain neurons of transgenic mice.

Dopamine D4 receptor-deficient mice display cortical hyperexcitability.

Absence of dopamine D4 receptors results in enhanced reactivity to unconditioned, but not conditioned, fear.

Fast axonal transport of the proteasome complex depends on membrane interaction and molecular motor function.

Mouse sperm begin to undergo acrosomal exocytosis in the upper isthmus of the oviduct.

Hippocampal to basal forebrain transport of Mn is impaired by deletion of KLC1, a subunit of the conventional kinesin microtubule-based motor.

Tau Isoforms Imbalance Impairs the Axonal Transport of the Amyloid Precursor Protein in Human Neurons.

αSynuclein control of mitochondrial homeostasis in human-derived neurons is disrupted by mutations associated with Parkinson's disease.

Mitochondrial dynamics in Parkinson's disease: a role for α-synuclein?

Methods for Quantitative Analysis of Axonal Cargo Transport.

Proteasome stress leads to APP axonal transport defects by promoting its amyloidogenic processing in lysosomes.

Neuronal KIF5b deletion induces striatum-dependent locomotor impairments and defects in membrane presentation of dopamine D2 receptors.

Kinesin-1-mediated axonal transport of CB1 receptors is required for cannabinoid-dependent axonal growth and guidance.

Differential mitochondrial roles for α-synuclein in DRP1-dependent fission and PINK1/Parkin-mediated oxidation.

DYRK1A regulates the bidirectional axonal transport of APP in human-derived neurons.

Modeling Alzheimer's Disease Using Human Brain Organoids.

Mitochondrial vulnerability to oxidation in human brain organoids modelling Alzheimer's disease.

Tau reduction with artificial microRNAs modulates neuronal physiology and improves tauopathy phenotypes in mice.