Timothy R. Nurkiewicz

Nanotechnology: toxicologic pathology.

Regulation of ion channels by integrins.

Particulate matter exposure impairs systemic microvascular endothelium-dependent dilation.

Calcium-independent release of endothelial nitric oxide in the arteriolar network: onset during rapid juvenile growth.

Reduced arteriolar responses to skeletal muscle contraction after ingestion of a high salt diet.

Systemic microvascular dysfunction and inflammation after pulmonary particulate matter exposure.

High salt intake reduces endothelium-dependent dilation of mouse arterioles via superoxide anion generated from nitric oxide synthase.

Hospitalization patterns associated with Appalachian coal mining.

Nanoparticle inhalation augments particle-dependent systemic microvascular dysfunction.

Age, gender, and hormonal status modulate the vascular toxicity of the diesel exhaust extract phenanthraquinone.

Pulmonary nanoparticle exposure disrupts systemic microvascular nitric oxide signaling.

Decreased arteriolar tetrahydrobiopterin is linked to superoxide generation from nitric oxide synthase in mice fed high salt.

Nanotoxicology--a pathologist's perspective.

Nanoparticle inhalation alters systemic arteriolar vasoreactivity through sympathetic and cyclooxygenase-mediated pathways.

Xenobiotic particle exposure and microvascular endpoints: a call to arms.

Changes in eNOS phosphorylation contribute to increased arteriolar NO release during juvenile growth.

Pulmonary particulate matter and systemic microvascular dysfunction.

Air pollution particulate matter collected from an appalachian mountaintop mining site induces microvascular dysfunction.

Impairment of coronary arteriolar endothelium-dependent dilation after multi-walled carbon nanotube inhalation: a time-course study.

Maternal Engineered Nanomaterial Exposure and Fetal Microvascular Function: Does the Barker Hypothesis Apply?

Pulmonary Cerium Dioxide Nanoparticle Exposure Differentially Impairs Coronary and Mesenteric Arteriolar Reactivity.