Manuel Zeitelhofer

High-efficiency transfection of mammalian neurons via nucleofection.

Transition from enhanced T cell infiltration to inflammation in the myelin-degenerative central nervous system.

Visualizing mRNA localization and local protein translation in neurons.

Dynamic interaction between P-bodies and transport ribonucleoprotein particles in dendrites of mature hippocampal neurons.

High-efficiency transfection of short hairpin RNAs-encoding plasmids into primary hippocampal neurons.

After injection into the striatum, in vitro-differentiated microglia- and bone marrow-derived dendritic cells can leave the central nervous system via the blood stream.

Perplexing bodies: The putative roles of P-bodies in neurons.

Transfection of cultured primary neurons via nucleofection.

Human cytomegalovirus infection is sensitive to the host cell DNA methylation state and alters global DNA methylation capacity.

Expression of Ccl11 associates with immune response modulation and protection against neuroinflammation in rats.

Imatinib ameliorates neuroinflammation in a rat model of multiple sclerosis by enhancing blood-brain barrier integrity and by modulating the peripheral immune response.

Efficacy of vitamin D in treating multiple sclerosis-like neuroinflammation depends on developmental stage.

Role of Tumor Pericytes in the Recruitment of Myeloid-Derived Suppressor Cells.

Presymptomatic activation of the PDGF-CC pathway accelerates onset of ALS neurodegeneration.

Expression of vascular endothelial growth factor (VEGF)-B and its receptor (VEGFR1) in murine heart, lung and kidney.

Lipocalin-2 as an Infection-Related Biomarker to Predict Clinical Outcome in Ischemic Stroke.

Functional genomics analysis of vitamin D effects on CD4+ T cells in vivo in experimental autoimmune encephalomyelitis ‬.

Widespread cortical demyelination of both hemispheres can be induced by injection of pro-inflammatory cytokines via an implanted catheter in the cortex of MOG-immunized rats.

Preclinical toxicological assessment of a novel monoclonal antibody targeting human platelet-derived growth factor CC (PDGF-CC) in PDGF-CChum mice.

Development of monoclonal anti-PDGF-CC antibodies as tools for investigating human tissue expression and for blocking PDGF-CC induced PDGFRα signalling in vivo.

Microglial autophagy-associated phagocytosis is essential for recovery from neuroinflammation.

Effect of Vitamin D on Experimental Autoimmune Neuroinflammation Is Dependent on Haplotypes Comprising Naturally Occurring Allelic Variants of CIITA ().

Blocking PDGF-CC signaling ameliorates multiple sclerosis-like neuroinflammation by inhibiting disruption of the blood-brain barrier.