Hamid I. Akbarali

Coupling of M2 muscarinic receptor to L-type Ca channel via c-src kinase in rabbit colonic circular smooth muscle.

Fenamate-induced enhancement of heterologously expressed HERG currents in Xenopus oocytes.

Cloning and functional characterization of the smooth muscle ether-a-go-go-related gene K+ channel. Potential role of a conserved amino acid substitution in the S4 region.

ERG K+ currents regulate pacemaker activity in ICC.

Altered gene expression and increased bursting activity of colonic smooth muscle ATP-sensitive K+ channels in experimental colitis.

Modulation of TRPV1 by nonreceptor tyrosine kinase, c-Src kinase.

Protein and gene expression of Ca2+ channel isoforms in murine colon: effect of inflammation.

Inflammation-induced "channelopathies" in the gastrointestinal smooth muscle.

Colonic inflammation increases Na+ currents in bladder sensory neurons.

Calcium carbonate antacids alter esophageal motility in heartburn sufferers.

Signal-transduction pathways that regulate smooth muscle function. II. Receptor-ion channel coupling mechanisms in gastrointestinal smooth muscle.

PKD2 functions as an epidermal growth factor-activated plasma membrane channel.

Cross-organ sensitization of lumbosacral spinal neurons receiving urinary bladder input in rats with inflamed colon.

The effects of sevoflurane and propofol on QT interval and heterologously expressed human ether-a-go-go related gene currents in Xenopus oocytes.

Nitrotyrosylation of Ca2+ channels prevents c-Src kinase regulation of colonic smooth muscle contractility in experimental colitis.

COOH-terminal association of human smooth muscle calcium channel Ca(v)1.2b with Src kinase protein binding domains: effect of nitrotyrosylation.

Denitration of L-type calcium channel.

Morphine tolerance in the mouse ileum and colon.

Colonic inflammation alters Src kinase-dependent gating properties of single Ca2+ channels via tyrosine nitration.

{alpha}7-nAChR-mediated suppression of hyperexcitability of colonic dorsal root ganglia neurons in experimental colitis.

Blockade of endocannabinoid hydrolytic enzymes attenuates precipitated opioid withdrawal symptoms in mice.

The role of β-arrestin2 in the mechanism of morphine tolerance in the mouse and guinea pig gastrointestinal tract.

Opioid-induced hypernociception is associated with hyperexcitability and altered tetrodotoxin-resistant Na+ channel function of dorsal root ganglia.

Nicotine suppresses hyperexcitability of colonic sensory neurons and visceral hypersensivity in mouse model of colonic inflammation.

Up-regulation of brain-derived neurotrophic factor in primary afferent pathway regulates colon-to-bladder cross-sensitization in rat.

Differences in the characteristics of tolerance to μ-opioid receptor agonists in the colon from wild type and β-arrestin2 knockout mice.

6β-N-heterocyclic substituted naltrexamine derivative NAP as a potential lead to develop peripheral mu opioid receptor selective antagonists.

Evidence for the putative cannabinoid receptor (GPR55)-mediated inhibitory effects on intestinal contractility in mice.

Morphine decreases enteric neuron excitability via inhibition of sodium channels.

Novel insights on the effect of nicotine in a murine colitis model.

Hydrogen sulfide as an allosteric modulator of ATP-sensitive potassium channels in colonic inflammation.

Design, synthesis, and biological evaluation of 17-cyclopropylmethyl-3,14β-dihydroxy-4,5α-epoxy-6β-[(4'-pyridyl)carboxamido]morphinan derivatives as peripheral selective μ opioid receptor Agents.

Electrophysiological characteristics of enteric neurons isolated from the immortomouse.

Sepiapterin ameliorates chemically-induced murine colitis and azoxymethane-induced colon cancer.