Juanita L. Merchant

An OmpA-like protein from Acinetobacter spp. stimulates gastrin and interleukin-8 promoters.

ZBP-89 promotes growth arrest through stabilization of p53.

VacA pores as portable portals for urea.

Genetic or chemical hypochlorhydria is associated with inflammation that modulates parietal and G-cell populations in mice.

A model for integrative study of human gastric acid secretion.

Regulation of epithelial cell growth by ZBP-89: potential relevance in pancreatic cancer.

Mutation of p53 in recurrent hepatocellular carcinoma and its association with the expression of ZBP-89.

Acinetobacter lwoffii infection and gastritis.

ZBP-89 mediates butyrate regulation of gene expression.

Treatment of Helicobacter gastritis with IL-4 requires somatostatin.

Transcription factor ZBP-89 is required for STAT1 constitutive expression.

Inflammation and cancer III. Somatostatin and the innate immune system.

Kinetic profiles of p300 occupancy in vivo predict common features of promoter structure and coactivator recruitment.

Helicobacter-induced intestinal metaplasia in the stomach correlates with Elk-1 and serum response factor induction of villin.

The Epstein-Barr virus protein BMRF1 activates gastrin transcription.

Modulating the cytokine response to treat Helicobacter gastritis.

Regulation and function of the sonic hedgehog signal transduction pathway in isolated gastric parietal cells.

Chronic gastritis in the hypochlorhydric gastrin-deficient mouse progresses to adenocarcinoma.

Interferon gamma induction of gastric mucous neck cell hypertrophy.

Inflammation, atrophy, gastric cancer: connecting the molecular dots.

Helicobacter pylori outer membrane protein 18 (Hp1125) induces dendritic cell maturation and function.

Somatostatin inhibits dendritic cell responsiveness to Helicobacter pylori.

Parietal cell hyperstimulation and autoimmune gastritis in cholera toxin transgenic mice.

Helicobacter pylori-secreted factors inhibit dendritic cell IL-12 secretion: a mechanism of ineffective host defense.

An isoform of ZBP-89 predisposes the colon to colitis.

Inhibition of growth hormone receptor gene expression by saturated fatty acids: role of Kruppel-like zinc finger factor, ZBP-89.

P53 mutants suppress ZBP-89 function.

Recruitment of ataxia-telangiectasia mutated to the p21(waf1) promoter by ZBP-89 plays a role in mucosal protection.

Intestinal overexpression of ZNF148 suppresses ApcMin/+ neoplasia.

Tales from the crypts: regulatory peptides and cytokines in gastrointestinal homeostasis and disease.

ATM phosphorylates ZBP-89 at Ser202 to potentiate p21waf1 induction by butyrate.

H pylori infection causes chronic pancreatitis in Mongolian gerbils.

Reduced pepsin A processing of sonic hedgehog in parietal cells precedes gastric atrophy and transformation.

Sumoylation-dependent control of homotypic and heterotypic synergy by the Kruppel-type zinc finger protein ZBP-89.

A role for CITED2, a CBP/p300 interacting protein, in colon cancer cell invasion.

Prospective identification of a multilineage progenitor in murine stomach epithelium.

What lurks beneath: IL-11, via Stat3, promotes inflammation-associated gastric tumorigenesis.

Regulated expression of the human gastrin gene in mice.

Hip1r is expressed in gastric parietal cells and is required for tubulovesicle formation and cell survival in mice.

Somatostatin stimulates menin gene expression by inhibiting protein kinase A.

Induction of follistatin precedes gastric transformation in gastrin deficient mice.

Sonic hedgehog in gastric physiology and neoplastic transformation: friend or foe?

ZBP-89 reduces the cell death threshold in hepatocellular carcinoma cells by increasing caspase-6 and S phase cell cycle arrest.

Paracrine Hedgehog signaling in stomach and intestine: new roles for hedgehog in gastrointestinal patterning.

Interleukin-1beta promotes gastric atrophy through suppression of Sonic Hedgehog.

Underrepresentation of underrepresented minorities in academic medicine: the need to enhance the pipeline and the pipe.

Hedgehog is an anti-inflammatory epithelial signal for the intestinal lamina propria.

Hedgehog signaling and gastrointestinal cancer.

Intracellular calcium release and protein kinase C activation stimulate sonic hedgehog gene expression during gastric acid secretion.

Bone morphogenetic protein signaling regulates gastric epithelial cell development and proliferation in mice.

ZBP-89 enhances Bak expression and causes apoptosis in hepatocellular carcinoma cells.

Hedgehog signaling in gastric physiology and cancer.

Helicobacter pylori induction of the gastrin promoter through GC-rich DNA elements.

Histamine 3 receptor activation mediates inhibition of acid secretion during Helicobacter-induced gastritis.

Gastric tuft cells express DCLK1 and are expanded in hyperplasia.

Menin and JunD regulate gastrin gene expression through proximal DNA elements.

Hedgehog signalling in gut development, physiology and cancer.

Interaction between ZBP-89 and p53 mutants and its contribution to effects of HDACi on hepatocellular carcinoma.

The same pocket in menin binds both MLL and JUND but has opposite effects on transcription.

Plasma Shh levels reduced in pancreatic cancer patients.

A high-fat diet regulates gastrin and acid secretion through primary cilia.

IFNγ contributes to the development of gastric epithelial cell metaplasia in Huntingtin interacting protein 1 related (Hip1r)-deficient mice.

Conditional deletion of menin results in antral G cell hyperplasia and hypergastrinemia.

Transgenic expression of interferon-γ in mouse stomach leads to inflammation, metaplasia, and dysplasia.

Butyrate increases IL-23 production by stimulated dendritic cells.

Inflammation and Gli2 suppress gastrin gene expression in a murine model of antral hyperplasia.

ZBP-89 regulates expression of tryptophan hydroxylase I and mucosal defense against Salmonella typhimurium in mice.

Gli1 deletion prevents Helicobacter-induced gastric metaplasia and expansion of myeloid cell subsets.

Dual oxidases control release of hydrogen peroxide by the gastric epithelium to prevent Helicobacter felis infection and inflammation in mice.

Epigenetic upregulation of Bak by ZBP-89 inhibits the growth of hepatocellular carcinoma.

Inhibition of Hedgehog signaling in the gastrointestinal tract: targeting the cancer microenvironment.

Identification of a functional TPH1 polymorphism associated with irritable bowel syndrome bowel habit subtypes.

Stress hematopoiesis is regulated by the Krüppel-like transcription factor ZBP-89.

Increased Expression of DUOX2 Is an Epithelial Response to Mucosal Dysbiosis Required for Immune Homeostasis in Mouse Intestine.

Invasive mouse gastric adenocarcinomas arising from Lgr5+ stem cells are dependent on crosstalk between the Hedgehog/GLI2 and mTOR pathways.

Deletion of and induces hypergastrinemia and gastric carcinoids.

SCHLAFEN 5 expression correlates with intestinal metaplasia that progresses to gastric cancer.

Schlafen 4-expressing myeloid-derived suppressor cells are induced during murine gastric metaplasia.

Recapitulating Human Gastric Cancer Pathogenesis: Experimental Models of Gastric Cancer.

Transcription Factor ZBP-89 Drives a Feedforward Loop of β-Catenin Expression in Colorectal Cancer.

NF-κB mediated transcription of DARPP-32 prevents -induced cell death.

Weight gain in mice on a high caloric diet and chronically treated with omeprazole depends on sex and genetic background.

Hedgehog Signaling Links Chronic Inflammation to Gastric Cancer Precursor Lesions.

Gastric Acid Secretion from Parietal Cells Is Mediated by a Ca Efflux Channel in the Tubulovesicle.

A Summary of the 2016 James W. Freston Conference of the American Gastroenterological Association: Intestinal Metaplasia in the Esophagus and Stomach: Origins, Differences, Similarities and Significance.

Pathophysiology of Gastric NETs: Role of Gastrin and Menin.

Gastrin Induces Nuclear Export and Proteasome Degradation of Menin in Enteric Glial Cells.

ZBP-89 function in colonic stem cells and during butyrate-induced senescence.

Indian Hedgehog Suppresses Intestinal Inflammation.

Parietal Cell Death by Cytokines.

Mature Enteroendocrine Cells Contributes to Basal and Pathological Stem Cell Dynamics in the Small Intestine.