Timothy J. LaRocca

Environmental pH modulates inerolysin activity via post-binding blockade.

Hyperglycemia potentiates a shift from apoptosis to RIP1-dependent necroptosis.

TRAIL (CD253) Sensitizes Human Airway Epithelial Cells to Toxin-Induced Cell Death.

Hyperglycemic Conditions Prime Cells for RIP1-dependent Necroptosis.

TNF Family Cytokines Induce Distinct Cell Death Modalities in the A549 Human Lung Epithelial Cell Line when Administered in Combination with Ricin Toxin.

Storage Primes Erythrocytes for Necroptosis and Clearance.

Sensitization of Airway Epithelial Cells to Toxin-Induced Death by TNF Superfamily Cytokines.

Mitochondrial ROS prime the hyperglycemic shift from apoptosis to necroptosis.

Necroptosis of Lung Epithelial Cells Triggered by Ricin Toxin and Bystander Inflammation.

Mitochondrial Trafficking of MLKL, Bak/Bax, and Drp1 Is Mediated by RIP1 and ROS which Leads to Decreased Mitochondrial Membrane Integrity during the Hyperglycemic Shift to Necroptosis.