Ryan S. Gray

A missense variant in SLC39A8 is associated with severe idiopathic scoliosis.

Mutations in Kinesin family member 6 reveal specific role in ependymal cell ciliogenesis and human neurological development.

Zebrafish: An Emerging Model for Orthopedic Research.

Regulation of terminal hypertrophic chondrocyte differentiation in mutant mice modeling infantile idiopathic scoliosis.

The Reissner Fiber Is Highly Dynamic In Vivo and Controls Morphogenesis of the Spine.

The developmental biology of kinesins.

Development of a straight vertebrate body axis.

Coding Variants Coupled With Rapid Modeling in Zebrafish Implicate Dynein Genes, and , as Adolescent Idiopathic Scoliosis Candidate Genes.

Postembryonic screen for mutations affecting spine development in zebrafish.

An adhesion G protein-coupled receptor is required in cartilaginous and dense connective tissues to maintain spine alignment.

The axonemal dynein heavy chain 10 gene is essential for monocilia motility and spine alignment in zebrafish.

Genetic animal modeling for idiopathic scoliosis research: history and considerations.

Kif9 is an active kinesin motor required for ciliary beating and proximodistal patterning of motile axonemes.