Adam R. Ferguson

Instrumental learning within the spinal cord. II. Evidence for central mediation.

Instrumental learning within the spinal cord: IV. Induction and retention of the behavioral deficit observed after noncontingent shock.

Instrumental learning within the spinal cord: V. Evidence the behavioral deficit observed after noncontingent nociceptive stimulation reflects an intraspinal modification.

GABA(A) receptor activation is involved in noncontingent shock inhibition of instrumental conditioning in spinal rats.

Brief exposure to a mild stressor enhances morphine-conditioned place preference in male rats.

The behavioral deficit observed following noncontingent shock in spinalized rats is prevented by the protein synthesis inhibitor cycloheximide.

A simple post hoc transformation that improves the metric properties of the BBB scale for rats with moderate to severe spinal cord injury.

Uncontrollable stimulation undermines recovery after spinal cord injury.

Instrumental learning within the rat spinal cord: localization of the essential neural circuit.

A sublethal dose of TNFalpha potentiates kainate-induced excitotoxicity in optic nerve oligodendrocytes.

Instrumental learning within the spinal cord: underlying mechanisms and implications for recovery after injury.

Exposure to intermittent nociceptive stimulation under pentobarbital anesthesia disrupts spinal cord function in rats.

The impact of morphine after a spinal cord injury.

Two chronic motor training paradigms differentially influence acute instrumental learning in spinally transected rats.

Developmental stage of oligodendrocytes determines their response to activated microglia in vitro.

Cell death after spinal cord injury is exacerbated by rapid TNF alpha-induced trafficking of GluR2-lacking AMPARs to the plasma membrane.

Group I metabotropic glutamate receptors control metaplasticity of spinal cord learning through a protein kinase C-dependent mechanism.

AMPA-receptor trafficking and injury-induced cell death.

Extensive spontaneous plasticity of corticospinal projections after primate spinal cord injury.

Quantitative CT Improves Outcome Prediction in Acute Traumatic Brain Injury.

Syndromics: A Bioinformatics Approach for Neurotrauma Research.

Methods for Functional Assessment After C7 Spinal Cord Hemisection in the Rhesus Monkey.