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Department of Pharmacology,
Vanderbilt Center for Bone Biology,
Department of Medicine,
Division of Clinical Pharmacology,
Department of Cancer Biology,
Department of Medicine, Division of Clinical Pharmacology
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Leptin regulates bone formation via the sympathetic nervous system.
Cell Nov, 2002 | Pubmed ID: 12419242
Common endocrine control of body weight, reproduction, and bone mass.
Annual review of nutrition , 2003 | Pubmed ID: 12730321
Reversible skeletal abnormalities in gamma-glutamyl transpeptidase-deficient mice.
Endocrinology Jul, 2003 | Pubmed ID: 12810527
[Control of body mass by leptin occurs through the sympathetic nervous system].
Médecine sciences : M/S Apr, 2003 | Pubmed ID: 12836204
Monosodium glutamate-sensitive hypothalamic neurons contribute to the control of bone mass.
Endocrinology Sep, 2003 | Pubmed ID: 12933656
Essential role of STAT3 in body weight and glucose homeostasis.
Molecular and cellular biology Jan, 2004 | Pubmed ID: 14673160
Enhanced bone formation in lipodystrophic PPARgamma(hyp/hyp) mice relocates haematopoiesis to the spleen.
EMBO reports Oct, 2004 | Pubmed ID: 15359271
Leptin regulation of bone resorption by the sympathetic nervous system and CART.
Nature Mar, 2005 | Pubmed ID: 15724149
Boning up on ephrin signaling.
Cell Aug, 2006 | Pubmed ID: 16901775
ATF4 mediation of NF1 functions in osteoblast reveals a nutritional basis for congenital skeletal dysplasiae.
Cell metabolism Dec, 2006 | Pubmed ID: 17141628
Regulation of bone remodeling by the central and peripheral nervous system.
Archives of biochemistry and biophysics May, 2008 | Pubmed ID: 18410742
Cocaine and amphetamine-regulated transcript may regulate bone remodeling as a circulating molecule.
Endocrinology Aug, 2008 | Pubmed ID: 18450969
Vimentin inhibits ATF4-mediated osteocalcin transcription and osteoblast differentiation.
The Journal of biological chemistry Oct, 2009 | Pubmed ID: 19726676
Skeletal abnormalities in neurofibromatosis type 1: approaches to therapeutic options.
American journal of medical genetics. Part A Oct, 2009 | Pubmed ID: 19764036
Atf4 regulates chondrocyte proliferation and differentiation during endochondral ossification by activating Ihh transcription.
Development (Cambridge, England) Dec, 2009 | Pubmed ID: 19906842
Local low-dose lovastatin delivery improves the bone-healing defect caused by Nf1 loss of function in osteoblasts.
Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research Jul, 2010 | Pubmed ID: 20200958
β2-Adrenergic receptor signaling in osteoblasts contributes to the catabolic effect of glucocorticoids on bone.
Endocrinology Apr, 2011 | Pubmed ID: 21266510
Mice lacking Nf1 in osteochondroprogenitor cells display skeletal dysplasia similar to patients with neurofibromatosis type I.
Human molecular genetics Oct, 2011 | Pubmed ID: 21757497
Genetic mouse models for bone studies--strengths and limitations.
Bone Dec, 2011 | Pubmed ID: 21907838
Chondrocytic Atf4 regulates osteoblast differentiation and function via Ihh.
Development (Cambridge, England) Feb, 2012 | Pubmed ID: 22190639
Stimulation of host bone marrow stromal cells by sympathetic nerves promotes breast cancer bone metastasis in mice.
PLoS biology Jul, 2012 | Pubmed ID: 22815651
Vanderbilt University
J. Preston Campbell1,2,
Alyssa R. Merkel2,3,4,
S. Kathryn Masood-Campbell2,4,
Florent Elefteriou1,2,4,5,
Julie A. Sterling2,3,4,5
1Department of Pharmacology, Vanderbilt University,
2Vanderbilt Center for Bone Biology, Vanderbilt University,
3Department of Veterans Affairs, Tennessee Valley Healthcare System (VISN 9),
4Department of Medicine, Division of Clinical Pharmacology, Vanderbilt University,
5Department of Cancer Biology, Vanderbilt University
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