Laboratory of Molecular Physiology,
National Institute on Alcohol Abuse and Alcoholism (NIAAA),
Laboratory of Molecular Physiology, National Institute on Alcohol Abuse and Alcoholism (NIAAA)
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The sources and sequestration of Ca(2+) contributing to neuroeffector Ca(2+) transients in the mouse vas deferens.
The Journal of physiology Dec, 2003 | Pubmed ID: 14500773
Inhibition of N-type calcium channels by activation of GPR35, an orphan receptor, heterologously expressed in rat sympathetic neurons.
The Journal of pharmacology and experimental therapeutics Jan, 2008 | Pubmed ID: 17940199
N-arachidonoyl L-serine, a putative endocannabinoid, alters the activation of N-type Ca2+ channels in sympathetic neurons.
Journal of neurophysiology Aug, 2008 | Pubmed ID: 18234973
Rapid modification of proteins using a rapamycin-inducible tobacco etch virus protease system.
PloS one , 2009 | Pubmed ID: 19830250
A Simple, Highly Efficient Method for Heterologous Expression in Mammalian Primary Neurons Using Cationic Lipid-mediated mRNA Transfection.
Frontiers in neuroscience , 2010 | Pubmed ID: 21267423
A functionally characterized test set of human induced pluripotent stem cells.
Nature biotechnology Mar, 2011 | Pubmed ID: 21293464
Modulation of neurite outgrowth by activation of calcium-permeable kainate receptors expressed by rat nociceptive-like dorsal root ganglion neurons.
Developmental neurobiology Oct, 2011 | Pubmed ID: 21557511
Mechanisms involved in nicotinic acetylcholine receptor-induced neurotransmitter release from sympathetic nerve terminals in the mouse vas deferens.
PloS one , 2011 | Pubmed ID: 22216213
National Institutes of Health (NIH)
Van B. Lu1,
Damian J. Williams1,
Yu-Jin Won1,
Stephen R. Ikeda1
1Laboratory of Molecular Physiology, National Institute on Alcohol Abuse and Alcoholism (NIAAA), National Institutes of Health (NIH)
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