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Laboratory of Molecular Physiology,
National Institute on Alcohol Abuse and Alcoholism (NIAAA),
Laboratory of Molecular Physiology, National Institute on Alcohol Abuse and Alcoholism (NIAAA)
Damian J. Williams has not added Biography.
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The sources and sequestration of Ca(2+) contributing to neuroeffector Ca(2+) transients in the mouse vas deferens.
The Journal of physiology Dec, 2003 | Pubmed ID: 14500773
Inhibition of N-type calcium channels by activation of GPR35, an orphan receptor, heterologously expressed in rat sympathetic neurons.
The Journal of pharmacology and experimental therapeutics Jan, 2008 | Pubmed ID: 17940199
N-arachidonoyl L-serine, a putative endocannabinoid, alters the activation of N-type Ca2+ channels in sympathetic neurons.
Journal of neurophysiology Aug, 2008 | Pubmed ID: 18234973
Rapid modification of proteins using a rapamycin-inducible tobacco etch virus protease system.
PloS one , 2009 | Pubmed ID: 19830250
A Simple, Highly Efficient Method for Heterologous Expression in Mammalian Primary Neurons Using Cationic Lipid-mediated mRNA Transfection.
Frontiers in neuroscience , 2010 | Pubmed ID: 21267423
A functionally characterized test set of human induced pluripotent stem cells.
Nature biotechnology Mar, 2011 | Pubmed ID: 21293464
Modulation of neurite outgrowth by activation of calcium-permeable kainate receptors expressed by rat nociceptive-like dorsal root ganglion neurons.
Developmental neurobiology Oct, 2011 | Pubmed ID: 21557511
Mechanisms involved in nicotinic acetylcholine receptor-induced neurotransmitter release from sympathetic nerve terminals in the mouse vas deferens.
PloS one , 2011 | Pubmed ID: 22216213
National Institutes of Health (NIH)
Van B. Lu1,
Damian J. Williams1,
Yu-Jin Won1,
Stephen R. Ikeda1
1Laboratory of Molecular Physiology, National Institute on Alcohol Abuse and Alcoholism (NIAAA), National Institutes of Health (NIH)
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