Stefan Bittner

The two-pore domain potassium channel TASK3 functionally impacts glioma cell death.

TWIK-related acid-sensitive K+ channel 1 (TASK1) and TASK3 critically influence T lymphocyte effector functions.

A beta-lactam antibiotic dampens excitotoxic inflammatory CNS damage in a mouse model of multiple sclerosis.

Altered neuronal expression of TASK1 and TASK3 potassium channels in rodent and human autoimmune CNS inflammation.

The neuroprotective impact of the leak potassium channel TASK1 on stroke development in mice.

TASK1 modulates inflammation and neurodegeneration in autoimmune inflammation of the central nervous system.

Comment on "Functional consequences of Kv1.3 ion channel rearrangement into the immunological synapse".

Collateral neuronal apoptosis in CNS gray matter during an oligodendrocyte-directed CD8(+) T cell attack.

Cytotoxic CD8+ T cell-neuron interactions: perforin-dependent electrical silencing precedes but is not causally linked to neuronal cell death.

Stromal interaction molecules 1 and 2 are key regulators of autoreactive T cell activation in murine autoimmune central nervous system inflammation.

Smad7 in T cells drives T helper 1 responses in multiple sclerosis and experimental autoimmune encephalomyelitis.

From the background to the spotlight: TASK channels in pathological conditions.

Upregulation of K2P5.1 potassium channels in multiple sclerosis.

Therapeutic approaches to multiple sclerosis: an update on failed, interrupted, or inconclusive trials of immunomodulatory treatment strategies.

Two pore domain potassium channels in cerebral ischemia: a focus on K2P9.1 (TASK3, KCNK9).

Therapeutic approaches to multiple sclerosis: an update on failed, interrupted, or inconclusive trials of neuroprotective and alternative treatment strategies.

Glatiramer acetate attenuates pro-inflammatory T cell responses but does not directly protect neurons from inflammatory cell death.

Natalizumab restores evoked potential abnormalities in patients with relapsing-remitting multiple sclerosis.

Active immunization with proteolipid protein (190-209) induces ascending paralysing experimental autoimmune encephalomyelitis in C3H/HeJ mice.

Blockade of the kinin receptor B1 protects from autoimmune CNS disease by reducing leukocyte trafficking.

Expression of K2P5.1 potassium channels on CD4+ T lymphocytes correlates with disease activity in rheumatoid arthritis patients.

Ion channels in autoimmune neurodegeneration.

Volume regulation of murine T lymphocytes relies on voltage-dependent and two-pore domain potassium channels.

IL-17 silencing does not protect nonobese diabetic mice from autoimmune diabetes.

CD4(+) CD25(+) FoxP3(+) regulatory T cells suppress cytotoxicity of CD8(+) effector T cells: implications for their capacity to limit inflammatory central nervous system damage at the parenchymal level.

The TASK1 channel inhibitor A293 shows efficacy in a mouse model of multiple sclerosis.

Identification of two-pore domain potassium channels as potent modulators of osmotic volume regulation in human T lymphocytes.

Regulatory T cells are strong promoters of acute ischemic stroke in mice by inducing dysfunction of the cerebral microvasculature.

Correction: Two pore domain potassium channels in cerebral ischemia: a focus on K2P9.1 (TASK3, KCNK9).

4-Aminopyridine ameliorates mobility but not disease course in an animal model of multiple sclerosis.

Rasmussen encephalitis treated with natalizumab.

Endothelial TWIK-related potassium channel-1 (TREK1) regulates immune-cell trafficking into the CNS.

Protein kinase Cβ as a therapeutic target stabilizing blood-brain barrier disruption in experimental autoimmune encephalomyelitis.

Targeting ion channels for the treatment of autoimmune neuroinflammation.

Excitotoxic neuronal cell death during an oligodendrocyte-directed CD8+ T cell attack in the CNS gray matter.

A splice variant of the two-pore domain potassium channel TREK-1 with only one pore domain reduces the surface expression of full-length TREK-1 channels.

CD4+NKG2D+ T cells exhibit enhanced migratory and encephalitogenic properties in neuroinflammation.

Evans syndrome associated with sterile inflammation of the central nervous system: a case report.

Ischemia-induced cell depolarization: does the hyperpolarization-activated cation channel HCN2 affect the outcome after stroke in mice?

TREK-King the Blood-Brain-Barrier.

Blocking of α4 integrin does not protect from acute ischemic stroke in mice.

Human CD4+ HLA-G+ regulatory T cells are potent suppressors of graft-versus-host disease in vivo.

Phospholipase D1 mediates lymphocyte adhesion and migration in experimental autoimmune encephalomyelitis.

Effects of glatiramer acetate in a spontaneous model of autoimmune neuroinflammation.

Three-dimensional organic microlasers with low lasing thresholds fabricated by multiphoton and UV lithography.

Transient receptor potential melastatin subfamily member 2 cation channel regulates detrimental immune cell invasion in ischemic stroke.

The two-pore domain potassium channel KCNK5 deteriorates outcome in ischemic neurodegeneration.

The CNS under pathophysiologic attack-examining the role of K2P channels.

Long-term efficacy of alemtuzumab in polymyositis.