Charles Krieger

Mitochondria, Ca2+ and neurodegenerative disease.

Protein phosphorylation networks in motor neuron death.

Lumbar motoneuron fate in a mouse model of amyotrophic lateral sclerosis.

Protein kinase and protein phosphatase expression in the central nervous system of G93A mSOD over-expressing mice.

Aberrant protein kinases and phosphoproteins in amyotrophic lateral sclerosis.

Chronically implanted epineural electrodes for repeated assessment of nerve conduction velocity and compound action potential amplitude in rodents.

Overexpressed mutant G93A superoxide dismutase protects calcineurin from inactivation.

Origin and distribution of bone marrow-derived cells in the central nervous system in a mouse model of amyotrophic lateral sclerosis.

Local self-renewal can sustain CNS microglia maintenance and function throughout adult life.

Neuromuscular dysfunction in the mutant superoxide dismutase mouse model of amyotrophic lateral sclerosis.

Pilot study of granulocyte colony stimulating factor (G-CSF)-mobilized peripheral blood stem cells in amyotrophic lateral sclerosis (ALS).

Bone marrow-derived cells in the central nervous system of a mouse model of amyotrophic lateral sclerosis are associated with blood vessels and express CX(3)CR1.

Mislocalization of TDP-43 in the G93A mutant SOD1 transgenic mouse model of ALS.

Fus gene mutations in familial and sporadic amyotrophic lateral sclerosis.

A second ALS patient having an L67P mutation in exon 3 of the Cu/Zn superoxide dismutase gene.

Drosophila adducin regulates Dlg phosphorylation and targeting of Dlg to the synapse and epithelial membrane.

Infiltrating monocytes trigger EAE progression, but do not contribute to the resident microglia pool.

Clinical and pathological features of amyotrophic lateral sclerosis caused by mutation in the C9ORF72 gene on chromosome 9p.

Reduced protein O-glycosylation in the nervous system of the mutant SOD1 transgenic mouse model of amyotrophic lateral sclerosis.

The Neuroinflammatory Response in ALS: The Roles of Microglia and T Cells.

Myelosuppressive conditioning using busulfan enables bone marrow cell accumulation in the spinal cord of a mouse model of amyotrophic lateral sclerosis.

Ethical considerations in the management of amyotrophic lateral sclerosis.

Activity-dependent secretion of progranulin from synapses.

Motoneuron firing in amyotrophic lateral sclerosis (ALS).

Phospho-regulated Drosophila adducin is a determinant of synaptic plasticity in a complex with Dlg and PIP2 at the larval neuromuscular junction.

Submyeloablative conditioning with busulfan permits bone marrow-derived cell accumulation in a murine model of Alzheimer's disease.