Peter K. Stys

Calpain-dependent neurofilament breakdown in anoxic and ischemic rat central axons.

Aberrant chloride transport contributes to anoxic/ischemic white matter injury.

Depolarization-induced Ca2+ release in ischemic spinal cord white matter involves L-type Ca2+ channel activation of ryanodine receptors.

Innervated human corneal equivalents as in vitro models for nerve-target cell interactions.

Nicotinic acetylcholine receptors in mouse and rat optic nerves.

White matter injury mechanisms.

Conversion of the Nikon C1 confocal laser-scanning head for multiphoton excitation on an upright microscope.

Axonal degeneration in multiple sclerosis: is it time for neuroprotective strategies?

Traumatic axonal injury induces proteolytic cleavage of the voltage-gated sodium channels modulated by tetrodotoxin and protease inhibitors.

Functional innervation in tissue engineered models for in vitro study and testing purposes.

General mechanisms of axonal damage and its prevention.

Spatiotemporal distribution of spectrin breakdown products induced by anoxia in adult rat optic nerve in vitro.

Na+-dependent sources of intra-axonal Ca2+ release in rat optic nerve during in vitro chemical ischemia.

Acute anterior circulation stroke: recanalization using clot angioplasty.

Complex interplay between glutamate receptors and intracellular Ca2+ stores during ischaemia in rat spinal cord white matter.

Sources of axonal calcium loading during in vitro ischemia of rat dorsal roots.

Real-time measurement of free Ca2+ changes in CNS myelin by two-photon microscopy.

Sodium channel blockers as neuroprotectants in neuroinflammatory disease: a double-edged sword.

White matter NMDA receptors: an unexpected new therapeutic target?

Effects of the noradrenergic system in rat white matter exposed to oxygen-glucose deprivation in vitro.

Glutamate receptors on myelinated spinal cord axons: II. AMPA and GluR5 receptors.

Glutamate receptors on myelinated spinal cord axons: I. GluR6 kainate receptors.

Virtual hypoxia and chronic necrosis of demyelinated axons in multiple sclerosis.

Gray matter pathology in (chronic) MS: modern views on an early observation.

Coherent anti-Stokes Raman scattering microscopy using photonic crystal fiber with two closely lying zero dispersion wavelengths.

Role of prions in neuroprotection and neurodegeneration: a mechanism involving glutamate receptors?

Degeneration versus autoimmunity in multiple sclerosis.

Mechanisms of axonal injury: internodal nanocomplexes and calcium deregulation.

Excitatory glycine responses of CNS myelin mediated by NR1/NR3 "NMDA" receptor subunits.

Miniaturized multimodal CARS microscope based on MEMS scanning and a single laser source.

Multiple sclerosis: autoimmune disease or autoimmune reaction?

The axo-myelinic synapse.

Aβ neurotoxicity depends on interactions between copper ions, prion protein, and N-methyl-D-aspartate receptors.

Copper-dependent regulation of NMDA receptors by cellular prion protein: implications for neurodegenerative disorders.

Metabolic injury to axons and myelin.

Will the real multiple sclerosis please stand up?

Sex-specific differences in retinal nerve fiber layer thinning after acute optic neuritis.

Pathoetiology of multiple sclerosis: are we barking up the wrong tree?

Treatment trials in progressive MS--current challenges and future directions.

High-resolution fluorescence microscopy of myelin without exogenous probes.

Skin-derived precursor Schwann cell myelination capacity in focal tibial demyelination.

Remyelination after spinal cord injury: is it a target for repair?

Patrolling monocytes play a critical role in CX3CR1-mediated neuroprotection during excitotoxicity.

White matter injury: Ischemic and nonischemic.

Editors' preface: the colourful white matter.

Cellular prion protein and NMDA receptor modulation: protecting against excitotoxicity.