Thomas Hoenen

VP40 octamers are essential for Ebola virus replication.

Ebola virus: unravelling pathogenesis to combat a deadly disease.

Infection of naive target cells with virus-like particles: implications for the function of ebola virus VP24.

In vitro evaluation of antisense RNA efficacy against filovirus infection, by use of reverse genetics.

Establishment and application of an infectious virus-like particle system for Marburg virus.

Efficient budding of the tacaribe virus matrix protein z requires the nucleoprotein.

Tacaribe virus but not junin virus infection induces cytokine release from primary human monocytes and macrophages.

Minigenomes, transcription and replication competent virus-like particles and beyond: reverse genetics systems for filoviruses and other negative stranded hemorrhagic fever viruses.

Genus-specific recruitment of filovirus ribonucleoprotein complexes into budding particles.

Ebola virus enters host cells by macropinocytosis and clathrin-mediated endocytosis.

Current ebola vaccines.

The Ebola virus glycoprotein contributes to but is not sufficient for virulence in vivo.

Inclusion bodies are a site of ebolavirus replication.

An upstream open reading frame modulates ebola virus polymerase translation and virus replication.

Phosphorylation of Ebola virus VP30 influences the composition of the viral nucleocapsid complex: impact on viral transcription and replication.

The role of oligomerization for the biological functions of the arenavirus nucleoprotein.

A novel Ebola virus expressing luciferase allows for rapid and quantitative testing of antivirals.

Ebola virus RNA editing depends on the primary editing site sequence and an upstream secondary structure.

Virus nomenclature below the species level: a standardized nomenclature for filovirus strains and variants rescued from cDNA.

Development and application of reporter-expressing mononegaviruses: current challenges and perspectives.

Ebola virus modulates transforming growth factor β signaling and cellular markers of mesenchyme-like transition in hepatocytes.

A Novel Life Cycle Modeling System for Ebola Virus Shows a Genome Length-Dependent Role of VP24 in Virus Infectivity.

Analysis of the highly diverse gene borders in Ebola virus reveals a distinct mechanism of transcriptional regulation.

Reverse genetics systems as tools for the development of novel therapies against filoviruses.

Lifecycle modelling systems support inosine monophosphate dehydrogenase (IMPDH) as a pro-viral factor and antiviral target for New World arenaviruses.

A genome-wide siRNA screen identifies a druggable host pathway essential for the Ebola virus life cycle.

Assessing cross-reactivity of Junín virus-directed neutralizing antibodies.