Lidia Hernandez

A progeroid syndrome in mice is caused by defects in A-type lamins.

The lipid phosphatase LPP3 regulates extra-embryonic vasculogenesis and axis patterning.

Paternal and maternal genomes confer opposite effects on proliferation, cell-cycle length, senescence, and tumor formation.

Mutations in the mouse Lmna gene causing progeria, muscular dystrophy and cardiomyopathy.

Preparation of acrylic bone cements for vertebroplasty with bismuth salicylate as radiopaque agent.

Influence of powder particle size distribution on complex viscosity and other properties of acrylic bone cement for vertebroplasty and kyphoplasty.

Loss of emerin at the nuclear envelope disrupts the Rb1/E2F and MyoD pathways during muscle regeneration.

New injectable and radiopaque antibiotic loaded acrylic bone cements.

Injectable acrylic bone cements for vertebroplasty based on a radiopaque hydroxyapatite. Bioactivity and biocompatibility.

Dysfunctional connections between the nucleus and the actin and microtubule networks in laminopathic models.

Functions of the nuclear envelope and lamina in development and disease.

Activation of NF-kappaB signaling by inhibitor of NF-kappaB kinase beta increases aggressiveness of ovarian cancer.

Nuclear factor kappaB transcription factors are coexpressed and convey a poor outcome in ovarian cancer.

Functional coupling between the extracellular matrix and nuclear lamina by Wnt signaling in progeria.

A mechanistic rationale for MEK inhibitor therapy in myeloma based on blockade of MAF oncogene expression.

IKK-ε coordinates invasion and metastasis of ovarian cancer.

Recent technological advances in using mouse models to study ovarian cancer.

Pathway-specific engineered mouse allograft models functionally recapitulate human serous epithelial ovarian cancer.

The small molecule NSC676914A is cytotoxic and differentially affects NFκB signaling in ovarian cancer cells and HEK293 cells.