Vincent Prinz

A medical humanities special study module on principles of medical theory and practice at the Charité, Humboldt University, Berlin, Germany.

TLR2 has a detrimental role in mouse transient focal cerebral ischemia.

Pituitary adenylate cyclase-activating polypeptide is up-regulated in cortical pyramidal cells after focal ischemia and protects neurons from mild hypoxic/ischemic damage.

The ectonucleotidase cd39/ENTPDase1 modulates purinergic-mediated microglial migration.

Intravenous rosuvastatin for acute stroke treatment: an animal study.

The acute (cerebro)vascular effects of statins.

Mrp-8 and -14 mediate CNS injury in focal cerebral ischemia.

CD93/AA4.1: a novel regulator of inflammation in murine focal cerebral ischemia.

Statins and stroke: prevention and beyond.

Heart rate contributes to the vascular effects of chronic mental stress: effects on endothelial function and ischemic brain injury in mice.

Stress worsens endothelial function and ischemic stroke via glucocorticoids.

Evidence of intrathecal immunoglobulin synthesis in stroke: a cohort study.

Exofocal dopaminergic degeneration as antidepressant target in mouse model of poststroke depression.

Multiple reimplantation technique for treatment of complex giant aneurysms of the middle cerebral artery: technical note.

The neurovascular unit as a selective barrier to polymorphonuclear granulocyte (PMN) infiltration into the brain after ischemic injury.

Membrane-type 1 metalloproteinase is upregulated in microglia/brain macrophages in neurodegenerative and neuroinflammatory diseases.

FLOW 800 allows visualization of hemodynamic changes after extracranial-to-intracranial bypass surgery but not assessment of quantitative perfusion or flow.

Influence of pigment epithelium-derived factor on outcome after striatal cerebral ischemia in the mouse.

The subpopulation of microglia expressing functional muscarinic acetylcholine receptors expands in stroke and Alzheimer's disease.

MRI heralds secondary nigral lesion after brain ischemia in mice: a secondary time window for neuroprotection.

High prevalence of pharmacologically induced platelet dysfunction in the acute setting of brain injury.

Autocrine release of angiopoietin-2 mediates cerebrovascular disintegration in Moyamoya disease.

Partial loss of VE-cadherin improves long-term outcome and cerebral blood flow after transient brain ischemia in mice.