Manel Camps

An rRNA mutation identifies the apicoplast as the target for clindamycin in Toxoplasma gondii.

Use of Pol I-deficient E. coli for functional complementation of DNA polymerase.

Targeted gene evolution in Escherichia coli using a highly error-prone DNA polymerase I.

When pol I goes into high gear: processive DNA synthesis by pol I in the cell.

Critical role of R-loops in processing replication blocks.

The BSR4 protein is up-regulated in Toxoplasma gondii bradyzoites, however the dominant surface antigen recognised by the P36 monoclonal antibody is SRS9.

Genetic constraints on protein evolution.

Modulation of ColE1-like plasmid replication for recombinant gene expression.

Targeted mutagenesis in E. coli: A powerful tool for the generation of random mutant libraries.

Roles of DNA polymerase I in leading and lagging-strand replication defined by a high-resolution mutation footprint of ColE1 plasmid replication.

Network models of TEM β-lactamase mutations coevolving under antibiotic selection show modular structure and anticipate evolutionary trajectories.

Unraveling a connection between DNA demethylation repair and cancer.

Quantifying plasmid copy number to investigate plasmid dosage effects associated with directed protein evolution.

Interplay between base excision repair activity and toxicity of 3-methyladenine DNA glycosylases in an E. coli complementation system.

Mechanisms of plasmid segregation: have multicopy plasmids been overlooked?

Mechanisms of Theta Plasmid Replication.

Fluorescence-Based Reporters for Detection of Mutagenesis in E. coli.

Network Analysis of Protein Adaptation: Modeling the Functional Impact of Multiple Mutations.

Genetic control of ColE1 plasmid stability that is independent of plasmid copy number regulation.

Genomic analysis of plasmid content in food isolates of E. coli strongly supports its role as a reservoir for the horizontal transfer of virulence and antibiotic resistance genes.