Darragh Duffy

CXCL10 antagonism and plasma sDPPIV correlate with increasing liver disease in chronic HCV genotype 4 infected patients.

The ABCs of viral hepatitis that define biomarker signatures of acute viral hepatitis.

Functional analysis via standardized whole-blood stimulation systems defines the boundaries of a healthy immune response to complex stimuli.

Apolipoprotein H expression is associated with IL28B genotype and viral clearance in hepatitis C virus infection.

The Milieu Intérieur study - an integrative approach for study of human immunological variance.

Semi-automated and standardized cytometric procedures for multi-panel and multi-parametric whole blood immunophenotyping.

Automated flow cytometric analysis across large numbers of samples and cell types.

Dynamic Changes of Post-Translationally Modified Forms of CXCL10 and Soluble DPP4 in HCV Subjects Receiving Interferon-Free Therapy.

Bystander hyperactivation of preimmune CD8+ T cells in chronic HCV patients.

Impact of IL28B, APOH and ITPA Polymorphisms on Efficacy and Safety of TVR- or BOC-Based Triple Therapy in Treatment-Experienced HCV-1 Patients with Compensated Cirrhosis from the ANRS CO20-CUPIC Study.

Protein biomarkers discriminate Leishmania major-infected and non-infected individuals in areas endemic for cutaneous leishmaniasis.

Inhibition of DPP4 activity in humans establishes its in vivo role in CXCL10 post-translational modification: prospective placebo-controlled clinical studies.

Efficacy of the Janus kinase 1/2 inhibitor ruxolitinib in the treatment of vasculopathy associated with TMEM173-activating mutations in 3 children.

Standardized Whole-Blood Transcriptional Profiling Enables the Deconvolution of Complex Induced Immune Responses.

Genetic Adaptation and Neandertal Admixture Shaped the Immune System of Human Populations.

Ultrasensitive HIV-1 p24 Assay Detects Single Infected Cells and Differences in Reservoir Induction by Latency Reversal Agents.

Detection of interferon alpha protein reveals differential levels and cellular sources in disease.

Brief Report: Blockade of TANK-Binding Kinase 1/IKKɛ Inhibits Mutant Stimulator of Interferon Genes (STING)-Mediated Inflammatory Responses in Human Peripheral Blood Mononuclear Cells.

Deconvolution of the Response to Bacillus Calmette-Guérin Reveals NF-κB-Induced Cytokines As Autocrine Mediators of Innate Immunity.

An in vitro diagnostic certified point of care single nucleotide test for IL28B polymorphisms.

Myogenic Progenitor Cells Exhibit Type I Interferon-Driven Proangiogenic Properties and Molecular Signature During Juvenile Dermatomyositis.

Standardized whole blood stimulation improves immunomonitoring of induced immune responses in multi-center study.

Type I interferon-mediated autoinflammation due to DNase II deficiency.

Distinctive roles of age, sex, and genetics in shaping transcriptional variation of human immune responses to microbial challenges.

Natural variation in the parameters of innate immune cells is preferentially driven by genetic factors.

T-cell biomarkers for diagnosis of tuberculosis: candidate evaluation by a simple whole blood assay for clinical translation.

Development and clinical validation of the Genedrive point-of-care test for qualitative detection of hepatitis C virus.

Milieu intérieur: Defining the boundaries of a healthy immune response for improved vaccination strategies.

Publisher Correction: Natural variation in the parameters of innate immune cells is preferentially driven by genetic factors.

JAK inhibitor improves type I interferon induced damage: proof of concept in dermatomyositis.

Development of a Bead-Based Multiplex Assay for the Analysis of the Serological Response against the Six Pathogens HAV, HBV, HCV, CMV, T. gondii, and H. pylori.

Study of the Humoral Immune Response towards HCV Genotype 4 Using a Bead-Based Multiplex Serological Assay.

Comment on: 'Aberrant tRNA processing causes an autoinflammatory syndrome responsive to TNF inhibitors' by Giannelou : mutations in result in a constitutive activation of type I interferon signalling.