Tyler K. Nygaard

The mechanism of direct heme transfer from the streptococcal cell surface protein Shp to HtsA of the HtsABC transporter.

Identification and characterization of the heme-binding proteins SeShp and SeHtsA of Streptococcus equi subspecies equi.

Community-associated methicillin-resistant Staphylococcus aureus skin infections: advances toward identifying the key virulence factors.

The SaeR/S gene regulatory system is essential for innate immune evasion by Staphylococcus aureus.

SaeR binds a consensus sequence within virulence gene promoters to advance USA300 pathogenesis.

Spectroscopic identification of heme axial ligands in HtsA that are involved in heme acquisition by Streptococcus pyogenes.

Characterization of a new cytotoxin that contributes to Staphylococcus aureus pathogenesis.

Alpha-toxin induces programmed cell death of human T cells, B cells, and monocytes during USA300 infection.

Staphylococcus aureus nuclease is an SaeRS-dependent virulence factor.

The role of innate immunity in promoting SaeR/S-mediated virulence in Staphylococcus aureus.

The impact of α-toxin on host cell plasma membrane permeability and cytokine expression during human blood infection by CA-MRSA USA300.

Bovine CCL28 Mediates Chemotaxis via CCR10 and Demonstrates Direct Antimicrobial Activity against Mastitis Causing Bacteria.

Interaction of Staphylococci with Human B cells.

Staphylococcus aureus SaeR/S-Regulated Factors Decrease Monocyte-Derived Tumor Necrosis Factor-α to Reduce Neutrophil Bactericidal Activity.

Aspartic Acid Residue 51 of SaeR Is Essential for Virulence.