Edward Perez-Reyes

Low-voltage-activated Ca2+ currents are generated by members of the CavT subunit family (alpha1G/H) in rat primary sensory neurons.

Differential distribution of three members of a gene family encoding low voltage-activated (T-type) calcium channels.

Functional properties of a new voltage-dependent calcium channel alpha(2)delta auxiliary subunit gene (CACNA2D2).

Ducky mouse phenotype of epilepsy and ataxia is associated with mutations in the Cacna2d2 gene and decreased calcium channel current in cerebellar Purkinje cells.

Gating kinetics of the alpha1I T-type calcium channel.

Stimulation of recombinant Ca(v)3.2, T-type, Ca(2+) channel currents by CaMKIIgamma(C).

Cloning and expression of the human T-type channel Ca(v)3.3: insights into prepulse facilitation.

Alternative splicing of the rat Ca(v)3.3 T-type calcium channel gene produces variants with distinct functional properties(1).

Molecular physiology of low-voltage-activated t-type calcium channels.

Cloning of a novel one-repeat calcium channel-like gene.

Paradoxical role of T-type calcium channels in coronary smooth muscle.

Functional impact of alternative splicing of human T-type Cav3.3 calcium channels.

The role of voltage gated T-type Ca2+ channel isoforms in mediating "capacitative" Ca2+ entry in cancer cells.

Contrasting anesthetic sensitivities of T-type Ca2+ channels of reticular thalamic neurons and recombinant Ca(v)3.3 channels.

Functional characterization and neuronal modeling of the effects of childhood absence epilepsy variants of CACNA1H, a T-type calcium channel.

Transfer of beta subunit regulation from high to low voltage-gated Ca2+ channels.

The endogenous redox agent L-cysteine induces T-type Ca2+ channel-dependent sensitization of a novel subpopulation of rat peripheral nociceptors.

CaV3.2 is the major molecular substrate for redox regulation of T-type Ca2+ channels in the rat and mouse thalamus.

Molecular characterization of T-type calcium channels.

Molecular biology of T-type calcium channels.

The I-II loop controls plasma membrane expression and gating of Ca(v)3.2 T-type Ca2+ channels: a paradigm for childhood absence epilepsy mutations.

Reducing agents sensitize C-type nociceptors by relieving high-affinity zinc inhibition of T-type calcium channels.

Molecular mechanisms of subtype-specific inhibition of neuronal T-type calcium channels by ascorbate.

Characterization of the gating brake in the I-II loop of Ca(v)3.2 T-type Ca(2+) channels.

I-II loop structural determinants in the gating and surface expression of low voltage-activated calcium channels.

Alternative splicing within the I-II loop controls surface expression of T-type Ca(v)3.1 calcium channels.

Orientation of the calcium channel beta relative to the alpha(1)2.2 subunit is critical for its regulation of channel activity.

Molecular pharmacology of human Cav3.2 T-type Ca2+ channels: block by antihypertensives, antiarrhythmics, and their analogs.

Molecular mechanisms of lipoic acid modulation of T-type calcium channels in pain pathway.

Orientation of palmitoylated CaVbeta2a relative to CaV2.2 is critical for slow pathway modulation of N-type Ca2+ current by tachykinin receptor activation.

Structural determinants of the high affinity extracellular zinc binding site on Cav3.2 T-type calcium channels.

Role of voltage-gated calcium channels in epilepsy.

T-type Ca(2+) channel modulation by otilonium bromide.

Characterization of the gating brake in the I-II loop of CaV3 T-type calcium channels.

The voltage dependence of gating currents of the neuronal CA(v)3.3 channel is determined by the gating brake in the I-II loop.

Enhanced actions of adenosine in medial entorhinal cortex layer II stellate neurons in temporal lobe epilepsy are mediated via A(1)-receptor activation.

Molecular and biophysical basis of glutamate and trace metal modulation of voltage-gated Ca(v)2.3 calcium channels.

Effects of eugenol on T-type Ca2+ channel isoforms.

Mechanisms by which a CACNA1H mutation in epilepsy patients increases seizure susceptibility.

A potassium leak channel silences hyperactive neurons and ameliorates status epilepticus.

Ins and outs of T-channel structure function.

PHYSIOLOGY. Regulation of breathing by CO₂ requires the proton-activated receptor GPR4 in retrotrapezoid nucleus neurons.

CaV3.2 calcium channels control NMDA receptor-mediated transmission: a new mechanism for absence epilepsy.

Contrasting the roles of the I-II loop gating brake in CaV3.1 and CaV3.3 calcium channels.

Activation of Pyramidal Neurons in Mouse Medial Prefrontal Cortex Enhances Food-Seeking Behavior While Reducing Impulsivity in the Absence of an Effect on Food Intake.

Central Mechanisms Mediating Thrombospondin-4-induced Pain States.

Nalcn Is a "Leak" Sodium Channel That Regulates Excitability of Brainstem Chemosensory Neurons and Breathing.

Ca Regulation of Ca3.3 T-type Ca Channel Is Mediated by Calmodulin.

Calmodulin regulates Ca3 T-type channels at their gating brake.

A novel therapeutic approach for treatment of catamenial epilepsy.

Functional variants in and may contribute to genetic generalized epilepsy.

CACHD1 is an α2δ-Like Protein That Modulates Ca3 Voltage-Gated Calcium Channel Activity.

LMO7 deficiency reveals the significance of the cuticular plate for hearing function.

Primary aldosteronism associated with a germline variant in .

Inhibition of T-Type calcium channels in mEC layer II stellate neurons reduces neuronal hyperexcitability associated with epilepsy.

Characterization of kindled VGAT-Cre mice as a new animal model of temporal lobe epilepsy.

A brainstem peptide system activated at birth protects postnatal breathing.

Effect of photobiomodulation on mitochondrial dynamics in peripheral nervous system in streptozotocin-induced type 1 diabetes in rats.