Youichi Shinozaki

Direct observation of ATP-induced conformational changes in single P2X(4) receptors.

The astrocyte-targeted therapy by Bushi for the neuropathic pain in mice.

In vitro blood-brain barrier models using brain capillary endothelial cells isolated from neonatal and adult rats retain age-related barrier properties.

Astrocytes protect neurons against methylmercury via ATP/P2Y(1) receptor-mediated pathways in astrocytes.

Microglia release ATP by exocytosis.

Microglia trigger astrocyte-mediated neuroprotection via purinergic gliotransmission.

Müller cell-mediated neurite outgrowth of the retinal ganglion cells via P2Y receptor signals.

Cortical astrocytes rewire somatosensory cortical circuits for peripheral neuropathic pain.

Urothelial ATP exocytosis: regulation of bladder compliance in the urine storage phase.

Transformation of Astrocytes to a Neuroprotective Phenotype by Microglia via P2Y Receptor Downregulation.

Polymorphic regulation of mitochondrial fission and fusion modifies phenotypes of microglia in neuroinflammation.

Purinergic dysregulation causes hypertensive glaucoma-like optic neuropathy.

The oscillation of intracellular Ca influx associated with the circadian expression of Piezo1 and TRPV4 in the bladder urothelium.

Anti-Depressant Fluoxetine Reveals its Therapeutic Effect Via Astrocytes.

The time-dependent variation of ATP release in mouse primary-cultured urothelial cells is regulated by the clock gene.

Microglia mediate non-cell-autonomous cell death of retinal ganglion cells.

Evaluation of M1-microglial activation by neurotoxic metals using optimized organotypic cerebral slice cultures.

Microglial ROCK is essential for chronic methylmercury-induced neurodegeneration.

Intermittent restraint stress induces circadian misalignment in the mouse bladder, leading to nocturia.

Author Correction: Intermittent restraint stress induces circadian misalignment in the mouse bladder, leading to nocturia.

[Pathogenic roles of retinal glia in glaucoma].

Potential roles of astrocytes and Müller cells in the pathogenesis of glaucoma.

Reactive astrocyte-driven epileptogenesis is induced by microglia initially activated following status epilepticus.

Loss of P2Y receptors triggers glaucoma-like pathology in mice.

Transnasal transplantation of human induced pluripotent stem cell-derived microglia to the brain of immunocompetent mice.

Adenosine A receptor down-regulates metabotropic glutamate receptor 5 in astrocytes during postnatal development.

The Mlc1 Promoter Directs Müller Cell-specific Gene Expression in the Retina.

Transient astrocytic mGluR5 expression drives synaptic plasticity and subsequent chronic pain in mice.