Jianyong Zhong

Expression of HIV-1 genes in podocytes alone can lead to the full spectrum of HIV-1-associated nephropathy.

HIV-1 genes vpr and nef synergistically damage podocytes, leading to glomerulosclerosis.

Induction of podocyte-derived VEGF ameliorates podocyte injury and subsequent abnormal glomerular development caused by puromycin aminonucleoside.

ARB protects podocytes from HIV-1 nephropathy independently of podocyte AT1.

Maturational regression of glomeruli determines the nephron population in normal mice.

Vitronectin-binding PAI-1 protects against the development of cardiac fibrosis through interaction with fibroblasts.

Effects of combination PPARγ agonist and angiotensin receptor blocker on glomerulosclerosis.

A perspective on chronic kidney disease progression.

Tubulointerstitial fibrosis can sensitize the kidney to subsequent glomerular injury.

Chronic kidney disease alters lipid trafficking and inflammatory responses in macrophages: effects of liver X receptor agonism.

Circulating α-Klotho is Related to Plasma Aldosterone and Its Follow-Up Change Predicts CKD Progression.

Cross Talk from Tubules to Glomeruli.

Kidney as modulator and target of "good/bad" HDL.

Modulation of aldosterone levels by aldosterone synthase promoter polymorphism and association with eGFR decline in patients with chronic kidney disease.

Dual inhibition of endothelial miR-92a-3p and miR-489-3p reduces renal injury-associated atherosclerosis.

Lipoprotein modulation of proteinuric renal injury.

Fibroblast-specific plasminogen activator inhibitor-1 depletion ameliorates renal interstitial fibrosis after unilateral ureteral obstruction.

Mechanisms of Scarring in Focal Segmental Glomerulosclerosis.

Urinary apolipoprotein AI in children with kidney disease.

Renal lymphatic vessel dynamics.

Stabilization of hypoxia-inducible factor ameliorates glomerular injury sensitization after tubulointerstitial injury.

Kidney injury-mediated disruption of intestinal lymphatics involves dicarbonyl-modified lipoproteins.

Biotic Supplements in Patients With Chronic Kidney Disease: Meta-Analysis of Randomized Controlled Trials.

Kidney Injury Causes Accumulation of Renal Sodium That Modulates Renal Lymphatic Dynamics.

Podocyte-Related Mechanisms Underlying Survival Benefit of Long-Term Angiotensin Receptor Blocker.

Dicarbonyl-modified lipoproteins contribute to proteinuric kidney injury.

Low-Dose Erythropoietin Amplifies Beneficial Effects of Angiotensin II Blockade on Glomerulosclerosis.

Intestinal Lymphatic Dysfunction in Kidney Disease.