Federico Sesti

A potassium channel-MiRP complex controls neurosensory function in Caenorhabditis elegans.

Hyperpolarization moves S4 sensors inward to open MVP, a methanococcal voltage-gated potassium channel.

Pore- and state-dependent cadmium block of I(Ks) channels formed with MinK-55C and wild-type KCNQ1 subunits.

Single-walled carbon nanotubes are a new class of ion channel blockers.

N-Glycosylation-dependent block is a novel mechanism for drug-induced cardiac arrhythmia.

A family of K+ channel ancillary subunits regulate taste sensitivity in Caenorhabditis elegans.

MPS-1 is a K+ channel beta-subunit and a serine/threonine kinase.

An evolutionarily conserved family of accessory subunits of K+ channels.

A new mode of regulation of N-type inactivation in a Caenorhabditis elegans voltage-gated potassium channel.

An arrhythmia susceptibility gene in Caenorhabditis elegans.

Molecular mechanisms underlying KVS-1-MPS-1 complex assembly.

The antiproliferative role of ERG K+ channels in rat osteoblastic cells.

Multiple modes of a-type potassium current regulation.

Oxidation of a potassium channel causes progressive sensory function loss during aging.

Auto-phosphorylation of a voltage-gated K+ channel controls non-associative learning.

Toxicity induced enhanced extracellular matrix production in osteoblastic cells cultured on single-walled carbon nanotube networks.

Oxidation of potassium channels by ROS: a general mechanism of aging and neurodegeneration?

The chemical and physical characteristics of single-walled carbon nanotube film impact on osteoblastic cell response.

Toxic role of K+ channel oxidation in mammalian brain.

An evolutionarily conserved mode of modulation of Shaw-like K⁺ channels.

Molecular mechanisms underlying the apoptotic effect of KCNB1 K+ channel oxidation.

Delayed pharyngeal repolarization promotes abnormal calcium buildup in aging muscle.

A common copy number variation (CNV) polymorphism in the CNTNAP4 gene: association with aging in females.

Oxidation of KCNB1 K(+) channels in central nervous system and beyond.

Guanine nucleotide exchange factor OSG-1 confers functional aging via dysregulated Rho signaling in Caenorhabditis elegans neurons.

Oxidation of ion channels in the aging nervous system.

Oxidation of K(+) Channels in Aging and Neurodegeneration.

Oxidation of KCNB1 Potassium Channels Causes Neurotoxicity and Cognitive Impairment in a Mouse Model of Traumatic Brain Injury.

Oxidation of KCNB1 potassium channels triggers apoptotic integrin signaling in the brain.

Cytoskeletal remodeling via Rho GTPases during oxidative and thermal stress in Caenorhabditis elegans.

Oxidation of KCNB1 channels in the human brain and in mouse model of Alzheimer's disease.

Oxidation of KCNB1 potassium channels in the murine brain during aging is associated with cognitive impairment.

Correction: Oxidation of KCNB1 potassium channels triggers apoptotic integrin signaling in the brain.

Complexes formed with integrin-α5 and KCNB1 potassium channel wild type or epilepsy-susceptibility variants modulate cellular plasticity Ras and Akt signaling.

A novel assay for drug screening that utilizes the heat shock response of Caenorhabditis elegans nematodes.

Antagonistic roles of Ras-MAPK and Akt signaling in integrin-K channel complex-mediated cellular apoptosis.

Non-conducting functions of ion channels: The case of integrin-ion channel complexes.

Integrin-KCNB1 potassium channel complexes regulate neocortical neuronal development and are implicated in epilepsy.

Corrigendum "Oxidation of KCNB1 potassium channels in the murine brain during aging is associated with cognitive impairment" [Biochem. Biophys. Res. Commun. 512(4) (2019) 665-669].

Apoptotic cell death in disease-Current understanding of the NCCD 2023.

Ion channels in neurodevelopment: lessons from the Integrin-KCNB1 channel complex.