Catherine A. Collins

Coordinating synaptic growth without being a nervous wreck.

Increased expression of the Drosophila vesicular glutamate transporter leads to excess glutamate release and a compensatory decrease in quantal content.

Highwire function at the Drosophila neuromuscular junction: spatial, structural, and temporal requirements.

A single vesicular glutamate transporter is sufficient to fill a synaptic vesicle.

Highwire restrains synaptic growth by attenuating a MAP kinase signal.

Synaptic development: insights from Drosophila.

Control of a kinesin-cargo linkage mechanism by JNK pathway kinases.

Visualizing glutamatergic cell bodies and synapses in Drosophila larval and adult CNS.

Protein turnover of the Wallenda/DLK kinase regulates a retrograde response to axonal injury.

A conditioning lesion protects axons from degeneration via the Wallenda/DLK MAP kinase signaling cascade.

Activation of Hsp70 reduces neurotoxicity by promoting polyglutamine protein degradation.

The Highwire ubiquitin ligase promotes axonal degeneration by tuning levels of Nmnat protein.

Bimodal control of dendritic and axonal growth by the dual leucine zipper kinase pathway.

Independent pathways downstream of the Wnd/DLK MAPKKK regulate synaptic structure, axonal transport, and injury signaling.

New approaches for studying synaptic development, function, and plasticity using Drosophila as a model system.

Sodium and potassium currents influence Wallerian degeneration of injured Drosophila axons.