Ari Barzilai

Accumulation of DNA damage and reduced levels of nicotine adenine dinucleotide in the brains of Atm-deficient mice.

Anti-semaphorin 3A antibodies rescue retinal ganglion cells from cell death following optic nerve axotomy.

The molecular mechanisms of dopamine toxicity.

Contribution of the Atm protein to maintaining cellular homeostasis evidenced by continuous activation of the AP-1 pathway in Atm-deficient brains.

ATM deficiency and oxidative stress: a new dimension of defective response to DNA damage.

Mammalian meiosis involves DNA double-strand breaks with 3' overhangs.

Molecular mechanisms of selective dopaminergic neuronal death in Parkinson's disease.

Up-regulation of semaphorin expression in retina of glaucomatous rabbits.

DNA damage responses to oxidative stress.

CuZn-SOD deficiency, rather than overexpression, is associated with enhanced recovery and attenuated activation of NF-kappaB after brain trauma in mice.

Impaired genomic stability and increased oxidative stress exacerbate different features of Ataxia-telangiectasia.

The herpes simplex virus type 1 vhs-UL41 gene secures viral replication by temporarily evading apoptotic cellular response to infection: Vhs-UL41 activity might require interactions with elements of cellular mRNA degradation machinery.

Nuclear ataxia-telangiectasia mutated (ATM) mediates the cellular response to DNA double strand breaks in human neuron-like cells.

Immunosuppressive role of semaphorin-3A on T cell proliferation is mediated by inhibition of actin cytoskeleton reorganization.

Analysis of the ataxia telangiectasia mutated-mediated DNA damage response in murine cerebellar neurons.

Examination of cellular and molecular events associated with optic nerve axotomy.

The contribution of the DNA damage response to neuronal viability.

MRI evidence of white matter damage in a mouse model of Nijmegen breakage syndrome.

The neurological phenotype of ataxia-telangiectasia: solving a persistent puzzle.

The role of the DNA damage response in neuronal development, organization and maintenance.

Upregulation of Semaphorin 3A and the associated biochemical and cellular events in a rat model of retinal detachment.

Analysis of the relationships between ATM and the Rad54 paralogs involved in homologous recombination repair.

Conditional inactivation of the NBS1 gene in the mouse central nervous system leads to neurodegeneration and disorganization of the visual system.

DNA damage, neuronal and glial cell death and neurodegeneration.

Sema-3A indirectly disrupts the regeneration process of goldfish optic nerve after controlled injury.

Astrocyte dysfunction associated with cerebellar attrition in a Nijmegen breakage syndrome animal model.

Investigation of the functional link between ATM and NBS1 in the DNA damage response in the mouse cerebellum.

Reduced synchronization persistence in neural networks derived from atm-deficient mice.

The neuro-glial-vascular interrelations in genomic instability symptoms.

A role for vascular deficiency in retinal pathology in a mouse model of ataxia-telangiectasia.

Malfunctioning DNA damage response (DDR) leads to the degeneration of nigro-striatal pathway in mouse brain.

The role of the neuro-astro-vascular unit in the etiology of ataxia telangiectasia.

Studying the cerebellar DNA damage response in the tissue culture dish.

The interrelations between malfunctioning DNA damage response (DDR) and the functionality of the neuro-glio-vascular unit.

Genome maintenance in the nervous system; insight into the role of the DNA damage response in brain development and disease.

Clique of functional hubs orchestrates population bursts in developmentally regulated neural networks.