Alexander Staruschenko

Fluorescence resonance energy transfer analysis of subunit stoichiometry of the epithelial Na+ channel.

Ras activates the epithelial Na(+) channel through phosphoinositide 3-OH kinase signaling.

Rho small GTPases activate the epithelial Na(+) channel.

Epithelial Na+ channel subunit stoichiometry.

Actin cytoskeleton disassembly affects conductive properties of stretch-activated cation channels in leukaemia cells.

Ras couples phosphoinositide 3-OH kinase to the epithelial Na+ channel.

Identification of a functional phosphatidylinositol 3,4,5-trisphosphate binding site in the epithelial Na+ channel.

Regulation of epithelial Na+ channel activity by conserved serine/threonine switches within sorting signals.

Regulation of the epithelial Na+ channel (ENaC) by phosphatidylinositides.

Rapid translocation and insertion of the epithelial Na+ channel in response to RhoA signaling.

Magnesium permeation through mechanosensitive channels: single-current measurements.

Functional reconstitution of the human epithelial Na+ channel in a mammalian expression system.

Binding and direct activation of the epithelial Na+ channel (ENaC) by phosphatidylinositides.

Quantifying RhoA facilitated trafficking of the epithelial Na+ channel toward the plasma membrane with total internal reflection fluorescence-fluorescence recovery after photobleaching.

Acute regulation of the epithelial Na+ channel by phosphatidylinositide 3-OH kinase signaling in native collecting duct principal cells.

Subunit-dependent cadmium and nickel inhibition of acid-sensing ion channels.

Molecular determinants of PI(4,5)P2 and PI(3,4,5)P3 regulation of the epithelial Na+ channel.

Ion channel regulation by Ras, Rho, and Rab small GTPases.

Mechanisms of non-steroid anti-inflammatory drugs action on ASICs expressed in hippocampal interneurons.

Insight toward epithelial Na+ channel mechanism revealed by the acid-sensing ion channel 1 structure.

Regulation of ENaC expression at the cell surface by Rab11.

Intrinsic voltage dependence of the epithelial Na+ channel is masked by a conserved transmembrane domain tryptophan.

Muscarinic M1 modulation of acid-sensing ion channels.

Peroxisome proliferator-activated receptor gamma antagonists decrease Na+ transport via the epithelial Na+ channel.

EGF and its related growth factors mediate sodium transport in mpkCCDc14 cells via ErbB2 (neu/HER-2) receptor.

Intact cytoskeleton is required for small G protein dependent activation of the epithelial Na+ channel.

Contribution of TRPV1-TRPA1 interaction to the single channel properties of the TRPA1 channel.

Endothelin-1 inhibits the epithelial Na+ channel through betaPix/14-3-3/Nedd4-2.

Regulation of ENaC-Mediated Sodium Reabsorption by Peroxisome Proliferator-Activated Receptors.

The actin cytoskeleton and small G protein RhoA are not involved in flow-dependent activation of ENaC.

Epidermal growth factor-mediated proliferation and sodium transport in normal and PKD epithelial cells.

Novel role of Rac1/WAVE signaling mechanism in regulation of the epithelial Na+ channel.

NSAIDs acutely inhibit TRPC channels in freshly isolated rat glomeruli.

Cortical actin binding protein cortactin mediates ENaC activity via Arp2/3 complex.

Effects of cytochrome P-450 metabolites of arachidonic acid on the epithelial sodium channel (ENaC).

Angiotensin II increases activity of the epithelial Na+ channel (ENaC) in distal nephron additively to aldosterone.

Role of βPix in the Kidney.

ROS production as a common mechanism of ENaC regulation by EGF, insulin, and IGF-1.

Direct activation of ENaC by angiotensin II: recent advances and new insights.

Regulation of transport in the connecting tubule and cortical collecting duct.

G-protein signaling modulator 1 deficiency accelerates cystic disease in an orthologous mouse model of autosomal dominant polycystic kidney disease.

Recording ion channels in isolated, split-opened tubules.

Single-channel analysis of TRPC channels in the podocytes of freshly isolated Glomeruli.

Regulation of ENaC in mice lacking renal insulin receptors in the collecting duct.

Detection of endogenous substances with enzymatic microelectrode biosensors in the kidney.

Real-time electrochemical detection of ATP and H₂O₂ release in freshly isolated kidneys.

Deficiency of renal cortical EGF increases ENaC activity and contributes to salt-sensitive hypertension.

Epidermal growth factors in the kidney and relationship to hypertension.

Aldosterone-dependent trans-activation and epigenetic derepression of ENaC: where is the balance?

Orally active epoxyeicosatrienoic acid analog attenuates kidney injury in hypertensive Dahl salt-sensitive rat.

Direct inhibition of basolateral Kir4.1/5.1 and Kir4.1 channels in the cortical collecting duct by dopamine.

Arp2/3 complex inhibitors adversely affect actin cytoskeleton remodeling in the cultured murine kidney collecting duct M-1 cells.

Pharmacological characterization of the P2 receptors profile in the podocytes of the freshly isolated rat glomeruli.

PC and PKC: in vivo vs. in vitro.

Angiotensin II has acute effects on TRPC6 channels in podocytes of freshly isolated glomeruli.

Epoxyeicosatrienoic acid analogue lowers blood pressure through vasodilation and sodium channel inhibition.

To cleave or not to cleave: role of ADAM17 in cell proliferation in PKD.

Mutation of Plekha7 attenuates salt-sensitive hypertension in the rat.

Role of Rho GDP dissociation inhibitor α in control of epithelial sodium channel (ENaC)-mediated sodium reabsorption.

Impaired epithelial Na+ channel activity contributes to cystogenesis and development of autosomal recessive polycystic kidney disease in PCK rats.

Cross-talk between insulin and IGF-1 receptors in the cortical collecting duct principal cells: implication for ENaC-mediated Na+ reabsorption.

Acetylation stimulates the epithelial sodium channel by reducing its ubiquitination and degradation.

Inhibition of ENaC by endothelin-1.

TRPC6 channel as an emerging determinant of the podocyte injury susceptibility in kidney diseases.

S&R Foundation 15th Anniversary.