Krista M. Heinonen

T-cell protein tyrosine phosphatase deletion results in progressive systemic inflammatory disease.

Genetic ablation of protein tyrosine phosphatase 1B accelerates lymphomagenesis of p53-null mice through the regulation of B-cell development.

Protein tyrosine phosphatase 1B negatively regulates macrophage development through CSF-1 signaling.

Protein tyrosine phosphatase 1B in hematopoiesis.

TC-PTP-deficient bone marrow stromal cells fail to support normal B lymphopoiesis due to abnormal secretion of interferon-{gamma}.

The signaling protein Wnt4 enhances thymopoiesis and expands multipotent hematopoietic progenitors through beta-catenin-independent signaling.

Development and functional properties of thymic and extrathymic T lymphocytes.

Protein tyrosine phosphatases PTP-1B and TC-PTP play nonredundant roles in macrophage development and IFN-gamma signaling.

SMAD3 prevents graft-versus-host disease by restraining Th1 differentiation and granulocyte-mediated tissue damage.

Wnt4 enhances murine hematopoietic progenitor cell expansion through a planar cell polarity-like pathway.

Wnt4 regulates thymic cellularity through the expansion of thymic epithelial cells and early thymic progenitors.

Wnt4, a pleiotropic signal for controlling cell polarity, basement membrane integrity, and antimüllerian hormone expression during oocyte maturation in the female follicle.

Frizzled-6 Regulates Hematopoietic Stem/Progenitor Cell Survival and Self-Renewal.