Tammy-Claire Troy

Permeability barrier dysfunction in transgenic mice overexpressing claudin 6.

Overexpression of the calcium sensing receptor accelerates epidermal differentiation and permeability barrier formation in vivo.

Barriers built on claudins.

Immunolocalization in the epidermis.

Epidermal cell analysis by RT-PCR.

Commitment of embryonic stem cells to an epidermal cell fate and differentiation in vitro.

Delayed epidermal permeability barrier formation and hair follicle aberrations in Inv-Cldn6 mice.

Derivation of epidermal colony-forming progenitors from embryonic stem cell cultures.

Directing epidermal fate selection by a novel co-culture system.

Role of the Cldn6 cytoplasmic tail domain in membrane targeting and epidermal differentiation in vivo.

Human embryonic stem cells: isolation, maintenance, and differentiation.

The temporal and spatial expression of Claudins in epidermal development and the accelerated program of epidermal differentiation in K14-CaSR transgenic mice.

Claudin expression modulations reflect an injury response in the murine epidermis.

Claudin immunolocalization in neonatal mouse epithelial tissues.

The targeted overexpression of a Claudin mutant in the epidermis of transgenic mice elicits striking epidermal and hair follicle abnormalities.

Changes in the distribution pattern of Claudin tight junction proteins during the progression of mouse skin tumorigenesis.

Insights into the role of the calcium sensing receptor in epidermal differentiation in vivo.

Dermatitis and aging-related barrier dysfunction in transgenic mice overexpressing an epidermal-targeted claudin 6 tail deletion mutant.

Involucrin-claudin-6 tail deletion mutant (CDelta206) transgenic mice: a model of delayed epidermal permeability barrier formation and repair.

Re-assessing K15 as an epidermal stem cell marker.

Using high-throughput immunoblotting to identify proteins involved in the differentiation of ES cells along the hair follicle lineage in vitro.

Junctions gone bad: claudins and loss of the barrier in cancer.