Courtney Sulentic

2,3,7,8-Tetrachlorodibenzo-p-dioxin (TCDD) alters the regulation and posttranslational modification of p27kip1 in lipopolysaccharide-activated B cells.

2,3,7,8-tetrachlorodibenzo-p-dioxin, an exogenous modulator of the 3'alpha immunoglobulin heavy chain enhancer in the CH12.LX mouse cell line.

Interactions at a dioxin responsive element (DRE) and an overlapping kappaB site within the hs4 domain of the 3'alpha immunoglobulin heavy chain enhancer.

Transgenic expression of Spi-C impairs B-cell development and function by affecting genes associated with BCR signaling.

Diverse chemicals including aryl hydrocarbon receptor ligands modulate transcriptional activity of the 3'immunoglobulin heavy chain regulatory region.

The long winding road toward understanding the molecular mechanisms for B-cell suppression by 2,3,7,8-tetrachlorodibenzo-p-dioxin.

Hydrogen peroxide modulates immunoglobulin expression by targeting the 3'Igh regulatory region through an NFκB-dependent mechanism.

2,3,7,8-tetrachlorodibenzo-p-dioxin induces transcriptional activity of the human polymorphic hs1,2 enhancer of the 3'Igh regulatory region.

The Toxicology Education Summit: building the future of toxicology through education.

A dioxin response element in the multiple cloning site of the pGL3 luciferase reporter influences transcriptional activity.

Analysis of modulation of immunoglobulin gene expression.

Gold nanoparticles induce transcriptional activity of NF-κB in a B-lymphocyte cell line.

The aryl hydrocarbon receptor regulates an essential transcriptional element in the immunoglobulin heavy chain gene.

The effect of shear flow on nanoparticle agglomeration and deposition in in vitro dynamic flow models.

The AhR and NF-κB/Rel Proteins Mediate the Inhibitory Effect of 2,3,7,8-Tetrachlorodibenzo-p-Dioxin on the 3' Immunoglobulin Heavy Chain Regulatory Region.