Department of Medicine-Cardiology Division
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Reconstituted slow muscarinic inhibition of neuronal (Ca(v)1.2c) L-type Ca2+ channels.
Biophysical journal Dec, 2002 | Pubmed ID: 12496094
Mapping sites of potential proximity between the dihydropyridine receptor and RyR1 in muscle using a cyan fluorescent protein-yellow fluorescent protein tandem as a fluorescence resonance energy transfer probe.
The Journal of biological chemistry Oct, 2004 | Pubmed ID: 15280389
Neurokinin 1 receptors trigger overlapping stimulation and inhibition of CaV2.3 (R-type) calcium channels.
Molecular pharmacology Jan, 2007 | Pubmed ID: 17050807
Bridging the myoplasmic gap: recent developments in skeletal muscle excitation-contraction coupling.
Journal of muscle research and cell motility , 2007 | Pubmed ID: 17899404
The alpha(1S) III-IV loop influences 1,4-dihydropyridine receptor gating but is not directly involved in excitation-contraction coupling interactions with the type 1 ryanodine receptor.
The Journal of biological chemistry Aug, 2008 | Pubmed ID: 18556650
The monomeric G proteins AGS1 and Rhes selectively influence Galphai-dependent signaling to modulate N-type (CaV2.2) calcium channels.
American journal of physiology. Cell physiology Nov, 2008 | Pubmed ID: 18815223
The skeletal L-type Ca(2+) current is a major contributor to excitation-coupled Ca(2+) entry.
The Journal of general physiology Jan, 2009 | Pubmed ID: 19114636
Reassessing the molecular mechanism of beta-adrenergic stimulation of cardiac L-type Ca2+ current.
Channels (Austin, Tex.) May-Jun, 2009 | Pubmed ID: 19535897
Effects of inserting fluorescent proteins into the alpha1S II-III loop: insights into excitation-contraction coupling.
The Journal of general physiology Jul, 2009 | Pubmed ID: 19564426
The cardiac alpha(1C) subunit can support excitation-triggered Ca2+ entry in dysgenic and dyspedic myotubes.
Channels (Austin, Tex.) Jul-Aug, 2009 | Pubmed ID: 19625771
The 63rd Symposium of the Society of General Physiologists: "muscles" instead of squid at Woods Hole.
The Journal of general physiology Dec, 2009 | Pubmed ID: 19948997
A malignant hyperthermia-inducing mutation in RYR1 (R163C): consequent alterations in the functional properties of DHPR channels.
The Journal of general physiology Jun, 2010 | Pubmed ID: 20479108
A malignant hyperthermia-inducing mutation in RYR1 (R163C): alterations in Ca2+ entry, release, and retrograde signaling to the DHPR.
The Journal of general physiology Jun, 2010 | Pubmed ID: 20479110
Looking for answers to EC coupling's persistent questions.
The Journal of general physiology Jul, 2010 | Pubmed ID: 20584887
Properties of Na+ currents conducted by a skeletal muscle L-type Ca2+ channel pore mutant (SkEIIIK).
Channels (Austin, Tex.) May-Jun, 2011 | Pubmed ID: 21406961
A shortcut to a skeletal muscle DHPR knock-in?
The Journal of physiology Oct, 2011 | Pubmed ID: 21965631
Malignant hyperthermia susceptibility arising from altered resting coupling between the skeletal muscle L-type Ca2+ channel and the type 1 ryanodine receptor.
Proceedings of the National Academy of Sciences of the United States of America May, 2012 | Pubmed ID: 22547813
Triclosan impairs excitation-contraction coupling and Ca2+ dynamics in striated muscle.
Proceedings of the National Academy of Sciences of the United States of America Aug, 2012 | Pubmed ID: 22891308
Ca(V)1.1: The atypical prototypical voltage-gated Ca²⁺ channel.
Biochimica et biophysica acta Jul, 2013 | Pubmed ID: 22982493
Impaired gating of an L-Type Ca(2+) channel carrying a mutation linked to malignant hyperthermia.
Biophysical journal May, 2013 | Pubmed ID: 23663834
Depressed pacemaker activity of sinoatrial node myocytes contributes to the age-dependent decline in maximum heart rate.
Proceedings of the National Academy of Sciences of the United States of America Oct, 2013 | Pubmed ID: 24128759
RGK protein-mediated impairment of slow depolarization- dependent Ca2+ entry into developing myotubes.
Channels (Austin, Tex.) , 2014 | Pubmed ID: 24476902
Functional assessment of three Rem residues identified as critical for interactions with Ca(2+) channel β subunits.
Pflugers Archiv : European journal of physiology Nov, 2015 | Pubmed ID: 25771954
Apparent lack of physical or functional interaction between CaV1.1 and its distal C terminus.
The Journal of general physiology Apr, 2015 | Pubmed ID: 25779869
Rem uncouples excitation-contraction coupling in adult skeletal muscle fibers.
The Journal of general physiology Jul, 2015 | Pubmed ID: 26078055
Bridging the myoplasmic gap II: more recent advances in skeletal muscle excitation-contraction coupling.
The Journal of experimental biology Jan, 2016 | Pubmed ID: 26792328
Distinct Components of Retrograde Ca(V)1.1-RyR1 Coupling Revealed by a Lethal Mutation in RyR1.
Biophysical journal Feb, 2016 | Pubmed ID: 26910427
Defining the MO's of RGK proteins.
Channels (Austin, Tex.) Sep, 2016 | Pubmed ID: 27249780
Progressive impairment of CaV1.1 function in the skeletal muscle of mice expressing a mutant type 1 Cu/Zn superoxide dismutase (G93A) linked to amyotrophic lateral sclerosis.
Skeletal muscle , 2016 | Pubmed ID: 27340545
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